ALS (Lou Gehrig's Disease)

Lou Gehrig was known as The Gentleman of Baseball. He played for the NY Yankees in the '20s and '30s. He had Amyotrophic Lateral Sclerosis (ALS) and, because of his fame and courage in fighting it, the disease also became known as Lou Gehrig's Disease. They made a movie about his life and his famous speech telling his beloved fans he had to quit.

Here is his bio.

A Washington Post columnist recently reviewed a new book about Lou Gehrig. He included an interesting quote from the book, “Luckiest Man: The Life and Death of Lou Gehrig” by Jonathan Eig, explaining that Gehrig's untimely death at age 39 from ALS changed it from an almost-unknown disease to a near-universally known disease.

Lida Mattman's slides of ALS blood show very aggressive CWD-bacteria. Dr. Mattman suggested that the bacteria causing ALS multiply rapidly and are, therefore, very aggressive. It isn't known if that translates into their being resistant to the MP or readily killed. Or if they are easily killed, if they would release more endotoxins. All these unknowns mean that treating ALS with the MP will have to be done very carefully. It is best in these situations for Dr to discuss the actual case with Dr Marshall.

Related FAQ:

Is the Marshall Protocol an Applicable Treatment for my Disease?

I personally believe that ALL the Th1 diseases result from the same bacterial pathogenesis. This belief is grounded in my understanding of how the bacteria directly drive the phagocytic biochemistry, causing the Th1 cytokineAny of various protein molecules secreted by cells of the immune system that serve to regulate the immune system. release.

The problem I have with discussing applicability of the MP to ALS, (or MS, or Diabetes, or even Parkinsons), is our current lack of understanding of how serious the immune system reactions might be, and in what form they might become manifest.

Consequently I try to focus on the diseases which we know respond well, and expand the list very slowly. I am always looking to link up with physicians who have these patients in a supportive atmosphere, preferably where 24/7 care is available from, for example, a mother/wife trained as a nurse, so that we can start to explore the outer limits of this Th1 pathogenesis.

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There are so many competing forces pushing and tugging at ALS patients that it is hard for them to be compliant and just focus on their recovery. With ALS the physicians, and the community, know that the patient is dying, and will push Vitamin D and just about every other medication onto them in a futile attempt to slow the degradation. It is hard for the (sick) patient to focus on recovery in the face of such pressures.

All applicable diseases are treated in a similar fashion on the MP with a few exceptions. Because ALS usually has a rapid course, one should progress through the MP as rapidly as is tolerable. Neurological symptoms are targeted better by one antibiotic in the second phase so that may be a priority to use.

With more aggressive syndromes, like ALS, it becomes more important to quickly force down the 25-DThe vitamin D metabolite widely (and erroneously) considered best indicator of vitamin D "deficiency." Inactivates the Vitamin D Nuclear Receptor. Produced by hydroxylation of vitamin D3 in the liver. (we don't have a way of doing this yet) and of getting them onto the multiple antibiotic protocols ASAP. We don't really have a good way of doing that either. So there will be some small variation between optimal treatments, especially for the most serious Th1 syndromes.

Trevor Marshall, PhD

Sample PubMed cite¹

Authors: Koch, Arthur L. Source: Critical Reviews in Microbiology; Jul-Sep2003, Vol. 29 Issue 3, p215-221, 7p

Abstract: Recently, a number of diseases that had been thought previously to be caused by something other than an infectious agent are now known to be caused by bacteria. It now appears that it is not uncommon that bacteria, viruses, or fungi can cause diseases even when these organisms have not been detected or cultured. The most recent, well-publicized case is that of stomach ulcers; these are largely due to Helicobacter pylori infections. Here, the possibility is explored that amyotrophic lateral sclerosis (ALS) is caused by a cell wall-deficient microorganism.

Author Affiliations: 1Biology Department, Indiana University, Bloomington, IN 47405-6801, USA ISSN: 1040-841X Accession Number: 11262074

Minocycline Delays Disease Onset and Mortality in a Transgenic Model of ALS Neuropharmacology Vol.13 No 8 12 June 2002 Ludo Van Den Bosch, Petra Tilkin, Griet Lemmns and Wim Robberecht Laboratory of Neurology, Campus Gasthuisberg, Herestraat 49, B-3000, Leuven, Belium

Active patients

• Chris Mavo: getting started, see also progress thread

Last updated: January 3, 2009

Patient interviews

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