Table of Contents

Immune boosters

Various medications and supplements have been described as “immune boosters” - substances which are stated as activating the immune response.

There are several reasons why a Marshall ProtocolA curative medical treatment for chronic inflammatory disease. Based on the Marshall Pathogenesis. (MP) patient should be hesitant to use these substances:

  1. Very few MP patients need any kind of an increase in immune response. In fact, most patients need to slow their immune response.
  2. The molecular actions of immune boosters including the ways in which the substances interact with the nuclear receptorsIntracellular receptor proteins that bind to hydrophobic signal molecules (such as steroid and thyroid hormones) or intracellular metabolites and are thus activated to bind to specific DNA sequences which affect transcription. is relatively unknown. These substances may interfere with the MP medications.
  3. Many have limited evidence of efficacy.

Types of immune boosters

Glyconutrients

Some researchers have concluded that Borrelia burgdorferi use sugars as part of their metabolism, which would mean that consuming glyconutrients benefits bacteria.

An effort to determine which carbohydrates Borrelia burgdorferi consumes revealed that the organism utilizes the monosaccharides glucose, mannose and N-acetylglucosamine, as well as the disaccharides maltose and chitobiose.

Robert Bradford et al.1)

Whey proteins

Whey proteins and numerous growth factors that regulate insulin secretion, differentiation of intestine epithelium cells, and also tissue restoration, are priceless in stimulating the immune system.

Lactoferrin shows the most comprehensive pro-health properties: antioxidative, anticancer, immune stimulative and even chemopreventive. Also peptides and amino acids formed from casein and whey proteins possess immune stimulative activity. The most valuable proteins, i.e. lactoferrin, immune globulins, lactoperoxidase and lisozyme, together with bioactive peptides, are resistant to pepsin and trypsin activity. This is why they maintain their exceptional biological activity. 2)

Reasons for using immune booster

MP is a therapy to restore innate immune responseThe body's first line of defense against intracellular and other pathogens. According to the Marshall Pathogenesis the innate immune system becomes disabled as patients develop chronic disease.. Perhaps one in a thousand of those suffering “auto-immune” disorders, apparently lack an Innate response. In these cases, alternate immune boosters seem appropriate.

Primary immunodeficiency

If immunopathologyA temporary increase in disease symptoms experienced by Marshall Protocol patients that results from the release of cytokines and endotoxins as disease-causing bacteria are killed. does not occur when carefully following the MP over a period of a few years, discuss with your doctor the possibility of one of these disorders. 3) 4)

From https://encyclopedia.thefreedictionary.com/Primary+immunodeficiency:-

Most primary immunodeficiencies are genetic disorders; the majority are diagnosed in children under the age of one, although milder forms may not be recognized until adulthood. While there are over 100 recognized PIDs, most are very rare. About 1 in 500 people in the United States are born with a primary immunodeficiency.[1] Immune deficiencies can result in persistent or recurring infections, autoinflammatory disorders, tumors, and disorders of various organs. There are currently no cures for these conditions; treatment is palliative and consists of managing infections and boosting the immune system.

When no longer on MP it would still be advisable to avoid RF radiation, vitamin D supplementation, heavy metal contaminants and any other immune suppressants.

5) 6) 7) 8)

see also

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===== References =====

1)
Bradford, RW and Allen, HW. (2006). Townsend Letter: The Examiner of Alternative Medicine. Biochemistry of Lyme disease: Borrelia burgdorferi spirochete/cyst.
2)
Ambroziak A, Cichosz G. [Immune stimulative potency of milk proteins]. Pol Merkur Lekarski. 2014 Feb;36(212):133-6.
[PMID: 24720113]
3)
Lim MS, Elenitoba-Johnson KSJ. The molecular pathology of primary immunodeficiencies. J Mol Diagn. 2004 May;6(2):59-83. doi: 10.1016/S1525-1578(10)60493-X.
[PMID: 15096561] [PMCID: 1867474] [DOI: 10.1016/S1525-1578(10)60493-X]
4)
Elenitoba-Johnson KS, Jaffe ES. Lymphoproliferative disorders associated with congenital immunodeficiencies. Semin Diagn Pathol. 1997 Feb;14(1):35-47.
[PMID: 9044508]
5)
Cao JQ, Li JX, Wang YY, Zhu FC. [Progress in research of influence of gene polymorphisms on immune response]. Zhonghua Liu Xing Bing Xue Za Zhi. 2018 Nov 10;39(11):1515-1518. doi: 10.3760/cma.j.issn.0254-6450.2018.11.018.
[PMID: 30462964] [DOI: 10.3760/cma.j.issn.0254-6450.2018.11.018]
6)
Schenck K, Kiyono H. Innate and acquired immunity, cytokines, and genetic factors in relation to the mucosal immune system. Acta Odontol Scand. 2001 Jun;59(3):121-3. doi: 10.1080/000163501750266693.
[PMID: 11501879] [DOI: 10.1080/000163501750266693]
7)
Chen H, Chong W, Teng C, Yao Y, Wang X, Li X. The immune response-related mutational signatures and driver genes in non-small-cell lung cancer. Cancer Sci. 2019 Aug;110(8):2348-2356. doi: 10.1111/cas.14113. Epub 2019 Jul 23.
[PMID: 31222843] [PMCID: 6676111] [DOI: 10.1111/cas.14113]
8)
Kelsen JR, Sullivan KE. Inflammatory Bowel Disease in Primary Immunodeficiencies. Curr Allergy Asthma Rep. 2017 Aug;17(8):57. doi: 10.1007/s11882-017-0724-z.
[PMID: 28755025] [PMCID: 6953999] [DOI: 10.1007/s11882-017-0724-z]