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home:diseases:aging [01.26.2019] – [summary of Olmesartan research on aging] sallieqhome:diseases:aging [01.26.2019] – [Aging (senescence)] sallieq
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  [[https://immunityageing.biomedcentral.com/articles/10.1186/1742-4933-3-12/comments|Recovery from some of the diseases of aging]]  [[https://immunityageing.biomedcentral.com/articles/10.1186/1742-4933-3-12/comments|Recovery from some of the diseases of aging]]
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 +Inflammation, But Not Telomere Length, Predicts Successful Ageing at Extreme Old Age: A Longitudinal Study of Semi-supercentenarians.    (({{pubmed>long:26629551}})) 
  
 Health of the hypothalamus may be a controlling factor as demonstrated in a mouse study by Yalin Zhang et al <blockquote> Mechanistically, hypothalamic stem/progenitor cells contributed greatly to exosomal microRNAs (miRNAs) in the cerebrospinal fluid, and these exosomal miRNAs declined during ageing, whereas central treatment with healthy hypothalamic stem/progenitor cell-secreted exosomes led to the slowing of ageing.  [[http://www.nature.com/nature/journal/vaop/ncurrent/full/nature23282.html|Hypothalamic stem cells control ageing speed partly through exosomal miRNAs]] Health of the hypothalamus may be a controlling factor as demonstrated in a mouse study by Yalin Zhang et al <blockquote> Mechanistically, hypothalamic stem/progenitor cells contributed greatly to exosomal microRNAs (miRNAs) in the cerebrospinal fluid, and these exosomal miRNAs declined during ageing, whereas central treatment with healthy hypothalamic stem/progenitor cell-secreted exosomes led to the slowing of ageing.  [[http://www.nature.com/nature/journal/vaop/ncurrent/full/nature23282.html|Hypothalamic stem cells control ageing speed partly through exosomal miRNAs]]
home/diseases/aging.txt · Last modified: 09.14.2022 by 127.0.0.1
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