This shows you the differences between two versions of the page.
Both sides previous revisionPrevious revisionNext revision | Previous revision | ||
home:diseases:anxiety [03.11.2019] – [Management of symptoms] sallieq | home:diseases:anxiety [09.14.2022] (current) – external edit 127.0.0.1 | ||
---|---|---|---|
Line 10: | Line 10: | ||
Patients suffering from anxiety may wish to modify their dose of antibiotics or take an [[home: | Patients suffering from anxiety may wish to modify their dose of antibiotics or take an [[home: | ||
- | ===== Evidence of infectious cause ===== | ||
- | Mark Lyte of the Texas Tech University School of Pharmacy noticed that lab mice dosed with // | ||
- | |||
- | < | ||
- | |||
- | //**L.E. Goehler** et al.// | ||
- | |||
- | Further, Neufeld //et al.// showed that germ-free mice exhibited reduced anxiety-like behavior as well as significant neurochemical changes in the brain compared to specific-pathogen-free mice. (({{pubmed> | ||
- | |||
- | |||
- | |||
- | The frequency with which patients of certain Th1 diseases also experience anxiety(({{pubmed> | ||
- | |||
- | |||
- | < | ||
- | It is my opinion that early and prominent symptoms of Th1 disease are psychological which have been interpreted as anxiety, depression, insomnia, learning disabilities etc. These symptoms like physical ones are exacerbated during effective treatment of Th1 [diseases]. | ||
- | |||
- | By understanding this, one can lessen some of the impact of those symptoms, just as one can with the physical symptoms. Not knowing why one is ' | ||
- | |||
- | //**Greg Blaney, MD**// </ | ||
Line 58: | Line 38: | ||
- | Finally there' | + | Finally there' |
Line 104: | Line 84: | ||
autism, ADHD, depression, severe anxiety, chronic fatigue syndrome (CFS) | autism, ADHD, depression, severe anxiety, chronic fatigue syndrome (CFS) | ||
- | Read the [[http:// | + | Read the [[https:// |
< | < | ||
Line 121: | Line 101: | ||
Interviews of patients with other diseases are [[home: | Interviews of patients with other diseases are [[home: | ||
+ | ===== Evidence of infectious cause ===== | ||
+ | Mark Lyte of the Texas Tech University School of Pharmacy noticed that lab mice dosed with // | ||
+ | < | ||
+ | //**L.E. Goehler** et al.// | ||
+ | Further, Neufeld //et al.// showed that germ-free mice exhibited reduced anxiety-like behavior as well as significant neurochemical changes in the brain compared to specific-pathogen-free mice. (({{pmid> | ||
+ | The frequency with which patients of certain Th1 diseases also experience anxiety(({{pmid> | ||
+ | < | ||
+ | It is my opinion that early and prominent symptoms of Th1 disease are psychological which have been interpreted as anxiety, depression, insomnia, learning disabilities etc. These symptoms like physical ones are exacerbated during effective treatment of Th1 [diseases]. | ||
+ | By understanding this, one can lessen some of the impact of those symptoms, just as one can with the physical symptoms. Not knowing why one is ' | ||
+ | //**Greg Blaney, MD**// </ | ||
+ | ===== Research into various drug effects ===== | ||
{{tag> | {{tag> | ||
+ | |||
+ | Findings suggest that a low dose of THC produces subjective stress-relieving effects in line with those commonly reported among cannabis users, but that higher doses may non-specifically increase negative mood. (({{pmid> | ||
+ | |||
+ | Baseline symptoms of depression (but not anxiety or stress) appeared to be exacerbated across time/ | ||
+ | |||
+ | Perhaps most well-studied is the role the CeA plays in unconditioned and conditioned fear generation (Ciocchi et al., 2010, Li et al., 2013, Tye et al., 2011), fear extinction, and conditioned inhibition (Amano et al., 2010), as well as conditioned orienting responses to emotionally salient stimuli (El-Amamy and Holland, 2007, Groshek et al., 2005). | ||
+ | |||
+ | the mechanisms by which stress increases amygdala-dmPFC synaptic strength and generates anxiety-like behaviors are not well understood. Here, we show that the mouse basolateral amygdala (BLA)-prelimbic prefrontal cortex (plPFC) circuit is engaged by stress and activation of this pathway in anxiogenic. [[https:// | ||
+ | |||
+ | |||
+ | |||
+ | | ||
+ | < | ||
===== Notes and comments ===== | ===== Notes and comments ===== | ||
Line 148: | Line 151: | ||
* Legacy content | * Legacy content | ||
- | * http:// | + | * https:// |
- | * http:// | + | * https:// |
- | * http:// | + | * https:// |
Line 168: | Line 171: | ||
Bacteria’s presence or absence affected how the mice used certain brain chemicals and genes involved in brain development. Taken together, the results indicate that intestinal bacteria somehow shape the brain and make mice more anxious — or cautious, depending how you look at it, Pettersson says. | Bacteria’s presence or absence affected how the mice used certain brain chemicals and genes involved in brain development. Taken together, the results indicate that intestinal bacteria somehow shape the brain and make mice more anxious — or cautious, depending how you look at it, Pettersson says. | ||
</ | </ | ||
- | ===== References ===== | + | ===== References =====</ |