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home:diseases:cancer [03.13.2019] – [H] sallieqhome:diseases:cancer [05.16.2019] – [Other Immune Suppressants] sallieq
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   * **liver cancer** – It is "well established" that chronic infections with hepatitis B and C viruses (HBV, HCV) represent major risk factors for HCC.(({{pubmed>long:10710202}}))   * **liver cancer** – It is "well established" that chronic infections with hepatitis B and C viruses (HBV, HCV) represent major risk factors for HCC.(({{pubmed>long:10710202}}))
   * **lung cancer** – According to a 2011 meta-analysis, patients exposed to //C. pneumoniae// infection had a 48% greater risk for lung cancer risk, relative to those not exposed.(({{pubmed>long:21194924}}))   * **lung cancer** – According to a 2011 meta-analysis, patients exposed to //C. pneumoniae// infection had a 48% greater risk for lung cancer risk, relative to those not exposed.(({{pubmed>long:21194924}}))
-  * **prostate cancer** – In a 2005 culture-based study, Cohen found that //Propionibacterium acnes// in 35% of prostate samples of 34 consecutive patients tested.(({{pubmed>long:15879794}})) +  * **prostate cancer** – In a 2005 culture-based study, Cohen found that //Propionibacterium acnes// in 35% of prostate samples of 34 consecutive patients tested.(({{pubmed>long:15879794}})) 
 +  * **Table of cancers** with associated agents. [[https://bmccancer.biomedcentral.com/track/pdf/10.1186/s12885-017-3234-4|Infections and Cancer]]
  
  
 With the recent advent of high throughput sequencing studies, we can expect additional insights into how diseases like cancer are associated with shifts in microbial communities.  (({{pubmed>long:27470177}})) With the recent advent of high throughput sequencing studies, we can expect additional insights into how diseases like cancer are associated with shifts in microbial communities.  (({{pubmed>long:27470177}}))
  
-==== see also MPSS Discussion ====+==== see also M P Study Site ====
  
 [[https://www.marshallprotocol.com/forum39/16259.html|Study shows mechanism for Microbiome to cause Cancer]] [[https://www.marshallprotocol.com/forum39/16259.html|Study shows mechanism for Microbiome to cause Cancer]]
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-==== Are There Concerns About Supplementing with Vitamin D? ====+==== Are There Concerns About V.D Supplement ? ====
  
 There is a common misconception that vitamin D supplementation is safe at any reasonable level, or that if some is good, more may be better. There is a common misconception that vitamin D supplementation is safe at any reasonable level, or that if some is good, more may be better.
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 Vitamin D receptor-mediated skewed differentiation of macrophages initiates myelofibrosis and subsequent osteosclerosis. (({{pubmed>long:30718230}}))  Vitamin D receptor-mediated skewed differentiation of macrophages initiates myelofibrosis and subsequent osteosclerosis. (({{pubmed>long:30718230}})) 
 +
 +2014  a Cochrane meta-analysis that included 18 randomized clinical trials comparing vitamin D administration versus no intervention in healthy population found no difference regarding cancer incidence.  In randomised controlled trials, relative risks for all cause mortality were 0.89 (0.80 to 0.99) for vitamin D3 supplementation and 1.04 (0.97 to 1.11) for vitamin D2 supplementation. The effects observed for vitamin D3 supplementation remained unchanged when grouped by various characteristics. However, for vitamin D2 supplementation, increased risks of mortality were observed in studies with lower intervention doses and shorter average intervention periods.  (({{pubmed>long:    24690623}}))
  
 ====1,25-dihydroxyvitamin D ==== ====1,25-dihydroxyvitamin D ====
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-Women with hereditary breast cancer predispositions should avoid using their smartphones, tablets, and laptops at night.   (({{pubmed>long:    29456806}}))+Women with hereditary breast cancer predispositions should put their smartphones, tablets, and laptops into air-plane mode at all times when not in use.   (({{pubmed>long:    29456806}})) 
 + 
 +Glioblastomas and malignant gliomas are the most common primary malignant brain tumors, with an annual incidence of 5.26 per 100,000 population. These tumors are typically associated with a dismal prognosis and poor quality of life. Only radiation exposure and certain genetic syndromes are well-defined risk factors for malignant glioma.   (({{pubmed>long:    24193082}}))  
 + 
 +This compound has antiproliferative and antimigratory effects in squamous cell carcinoma, glioblastoma, and breast cancer cell lines (({{pubmed>long:    31025400}})) 
 + 
 +Several researchers have highlighted a possible link between glioma and human cytomegalovirus (HCMV).  (({{pubmed>long:30888562}}))  
 + 
 +[[https://sacramento.cbslocal.com/2019/03/25/ripon-cell-tower-school-removal-cancer/|Cell tower removed after 4 students get cancers]] 
 + 
 +[[https://prepforthat.com/portland-blocking-5g-networks-over-health-risks/|Officials Attempt To Block 5G Network Installation Over Health Risks]]
  
  
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 Alicia H. Chang and Julie Parsonnet of Stanford University writes that a transmissible cause of cancer was suspected as early as the 16th century. It was not until the late 20th century definitively identified a bacterial cause of at least some types of cancer to most researchers' satisfaction.(({{pubmed>long:20930075}})) Identifying the full range of microbes which cause cancer, however, has been more challenging. To date, there have been a handful of cases that fulfill Koch's postulates (e.g., //H. pylori// and gastric cancer) where a single microbe is known to cause a single type of cancer. Consistent with Koch, researchers have tended to work in this vein, for example, looking at the 500+ species that inhabit the colon in the hopes of pinpointing the one and only bacterial species responsible for colorectal cancer.(({{pubmed>long:20930075}})) Alicia H. Chang and Julie Parsonnet of Stanford University writes that a transmissible cause of cancer was suspected as early as the 16th century. It was not until the late 20th century definitively identified a bacterial cause of at least some types of cancer to most researchers' satisfaction.(({{pubmed>long:20930075}})) Identifying the full range of microbes which cause cancer, however, has been more challenging. To date, there have been a handful of cases that fulfill Koch's postulates (e.g., //H. pylori// and gastric cancer) where a single microbe is known to cause a single type of cancer. Consistent with Koch, researchers have tended to work in this vein, for example, looking at the 500+ species that inhabit the colon in the hopes of pinpointing the one and only bacterial species responsible for colorectal cancer.(({{pubmed>long:20930075}}))
  
-A number of types of chronic infection have been observed to be associated with cancers in some studies, but without an identified specific bacterial pathogen. Examples are chronic ulcers and osteomyelitis sinus tracts (squamous cell carcinoma), chronic urinary tract infections (UTIs) (bladder cancer), and chronic prostatitis (prostate cancer).(({{pubmed>long:20930075}}))+A number of types of chronic infection have been observed to be associated with cancers in some studies, but without an identified specific bacterial pathogen. Examples are chronic ulcers and osteomylitis, sinus tracts (squamous cell carcinoma), chronic urinary tract infections (UTIs) (bladder cancer), and chronic prostatitis (prostate cancer).(({{pubmed>long:20930075}}))
  
 The Marshall Pathogenesis is at complete odds with Koch's postulates, suggesting that it is not one species but entire communities of microbes that are responsible for many cancers. The process by which a person accumulates microbes responsible for disease is known as “successive infection.” In successive infection, an infectious cascade of pathogens progressively slow the immune response and allow for subsequent infections to proliferate, resulting in dysbiosis (microbial imbalances). According to this model, a single microbe may not need to be present in all cases of disease in order to be implicated in it. The Marshall Pathogenesis is at complete odds with Koch's postulates, suggesting that it is not one species but entire communities of microbes that are responsible for many cancers. The process by which a person accumulates microbes responsible for disease is known as “successive infection.” In successive infection, an infectious cascade of pathogens progressively slow the immune response and allow for subsequent infections to proliferate, resulting in dysbiosis (microbial imbalances). According to this model, a single microbe may not need to be present in all cases of disease in order to be implicated in it.
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  <blockquote>These findings suggest that pro-tumorigenic entotic engulfment activity is associated with mutant p53 expression, and the two combined are a key factor in genomic instability (({{pubmed>long:30076358}}))</blockquote>   <blockquote>These findings suggest that pro-tumorigenic entotic engulfment activity is associated with mutant p53 expression, and the two combined are a key factor in genomic instability (({{pubmed>long:30076358}}))</blockquote> 
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-==== Recent research ====+==== Research ====
  
 This result provides a new idea on the anti-tumor mechanism of olmesartan, which may be used as a novel therapeutic target of cervical cancer.  (({{pubmed>long:28304186}})) This result provides a new idea on the anti-tumor mechanism of olmesartan, which may be used as a novel therapeutic target of cervical cancer.  (({{pubmed>long:28304186}}))
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 =====Other treatments===== =====Other treatments=====
  
-====Vitamin D======== 
-<mainarticle> [[home:pathogenesis:vitamind:cancer|Vitamin D and cancer]] </article> 
  
-{{section>:home:pathogenesis:vitamind:cancer#vitamin_D_and_cancer&noheader&firstseconly}}+[[http://cancerres.aacrjournals.org/content/69/6/2260.short|Bicarbonate Increases Tumor pH and Inhibits Spontaneous Metastases]]
  
 ====Chemotherapy and radiation==== ====Chemotherapy and radiation====
  
-Chemotherapy is aggressively immunosuppressive, and will allow a patient's bacterial load to return, by shutting down your immune system. For example, hepatitis infections(({{pubmed>long:21472116}})) as is pneumocystis pneumonia have been known to become reactivated during chemotherapy.(({{pubmed>long:17458875}})) +Chemotherapy is aggressively immunosuppressive, and will allow a patient's bacterial load to return, by shutting down your immune system. For example, hepatitis infections(({{pubmed>long:21472116}})); also pneumocystis pneumonia have been known to become reactivated during chemotherapy.(({{pubmed>long:17458875}}))  
 +==== Treatment for acute active Cancers====
  
  
 +[[https://www.nature.com/articles/nrc3298|Evolutionary dynamics of carcinogenesis and why targeted therapy does not work]]
  
 +[[http://cancerres.aacrjournals.org/content/69/11/4894.short|Adaptive Therapy]]
  
 +==== Toll-like receptor 9 (TLR9) agonists ====
 +
 +The activity and safety of these novel anticancer agents are being explored in a wide range of tumor types as part of a variety of therapeutic strategies with the goal of harnessing the immune response to fight cancer.  (({{pubmed>long:18176597}})) 
 +
 +Activation of Toll-Like Receptor 9 by DNA from Different Bacterial Species   (({{pubmed>long:16428738}})) 
 ====Vaccines==== ====Vaccines====
  
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 //**Gene**, MarshallProtocol.com//</blockquote>  //**Gene**, MarshallProtocol.com//</blockquote> 
 +==== Vitamin D====
 +<mainarticle> [[home:pathogenesis:vitamind:cancer|Vitamin D and cancer]] </article>
 +
 +{{section>:home:pathogenesis:vitamind:cancer#vitamin_D_and_cancer&noheader&firstseconly}}
 +
 +
 +=== Vitamin E ===
 +
 +
 +Vitamin E supplementation and the risk of heart failure in women.
 +(({{pubmed>long:    22438520}}))
 +
 +Supplementation with vitamin E did not result in any significant improvements in prognostic or functional indexes of heart failure or in the quality of life of patients with advanced heart failure.(({{pubmed>long:    11157316}}))
 +
 +Canolol was found to counteract the fibrotic effects of vitamin E + CoQ10 on cardiac fibrosis in the context of a high-fat diet enriched with RSO. This effect occurred through a restoration of cardiac Ag2R-1b mRNA expression and decreased ischemia.(({{pubmed>long:    29456586}}))
 +
 +
  
  
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 Researchers trialling gene therapies against cancer have been unsuccessful to date. Such initiatives have yet to take into account the types of intracellular microbes implicated by the Marshall Pathogenesis. One, a Duke University program, to tailor cancer treatments to certain cancers [[http://nyti.ms/q2fXgK|has ended in disaster and lawsuits]].  Researchers trialling gene therapies against cancer have been unsuccessful to date. Such initiatives have yet to take into account the types of intracellular microbes implicated by the Marshall Pathogenesis. One, a Duke University program, to tailor cancer treatments to certain cancers [[http://nyti.ms/q2fXgK|has ended in disaster and lawsuits]]. 
  
-==== Toll-like receptor 9 (TLR9) agonists ==== 
  
-The activity and safety of these novel anticancer agents are being explored in a wide range of tumor types as part of a variety of therapeutic strategies with the goal of harnessing the immune response to fight cancer.  (({{pubmed>long:18176597}}))  
- 
-Activation of Toll-Like Receptor 9 by DNA from Different Bacterial Species   (({{pubmed>long:16428738}}))  
 =====Screening and diagnosis=====   =====Screening and diagnosis=====  
  
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 ===== Recent research ===== ===== Recent research =====
- 
  
 Paclitaxel Reduces Tumor Growth by Reprogramming Tumor-Associated Macrophages to an M1 Profile in a TLR4-Dependent Manner.  (({{pubmed>long:30104241}}))  Paclitaxel Reduces Tumor Growth by Reprogramming Tumor-Associated Macrophages to an M1 Profile in a TLR4-Dependent Manner.  (({{pubmed>long:30104241}})) 
- 
  
 {{section>:home:pathogenesis:innate_immunity#recent_research_into_valuable_proteins&noheader}} {{section>:home:pathogenesis:innate_immunity#recent_research_into_valuable_proteins&noheader}}
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 miR-205 mediates the inhibition of cervical cancer cell proliferation using olmesartan.  (({{pubmed>long:    28304186}})) miR-205 mediates the inhibition of cervical cancer cell proliferation using olmesartan.  (({{pubmed>long:    28304186}}))
 +
 +Therapeutic potentials of SCFA receptors [section 7 in   (({{pubmed>long:27113407}})) ]
 +Recent progress in understanding the role of GPR41, GPR43 and GPR109A in health and disease demonstrate that these receptors do not just regulate inflammation and cancer in intestine, but also influence other gastrointestinal functions, allergies, adipogenesis, central nervous system and cardiovascular health.
 +
  
  
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   * [[https://en.wikipedia.org/wiki/List_of_unproven_and_disproven_cancer_treatments|Wiki list of unproven & disproven treatments]]   * [[https://en.wikipedia.org/wiki/List_of_unproven_and_disproven_cancer_treatments|Wiki list of unproven & disproven treatments]]
   * [[https://www.amjmed.com/article/S0002-9343(15)00509-4/pdf|Questions About Vitamin D for Primary Care Practice: Input From an NIH Conference]]   * [[https://www.amjmed.com/article/S0002-9343(15)00509-4/pdf|Questions About Vitamin D for Primary Care Practice: Input From an NIH Conference]]
-  * [[http://www.foodsmatter.com/es/health_risks/articles/goldsworthy-biological-effects-04-12.pdf|biological effects]]+  * [[http://www.foodsmatter.com/es/health_risks/articles/goldsworthy-biological-effects-04-12.pdf|biological effects of non-ionising electromagnetic fields]]
   * [[http://bioinitiative.org/report/wp-content/uploads/pdfs/sec01_2012_summary_for_public.pdf|Bioinitiative Report]]   * [[http://bioinitiative.org/report/wp-content/uploads/pdfs/sec01_2012_summary_for_public.pdf|Bioinitiative Report]]
  
home/diseases/cancer.txt · Last modified: 09.14.2022 by 127.0.0.1
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