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home:diseases:cardiovascular [05.24.2019] – [More information] sallieqhome:diseases:cardiovascular [04.09.2020] – [Other evidence] sallieq
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   * **[[home:pathogenesis:evolution|evolutionary analysis]] suggests cardiovascular disease is not caused by lifestyle or genetic factors** – Atherosclerosis has been identified in Egyptian mummies.(({{pubmed>long:10495769}})) This suggests that whatever factors cause atherosclerosis has existed for at least several millenia. If it were true that a factor such as a harmful allele for a gene or the consumption of fatty foods caused atherosclerosis, the human species would have had several millenia to evolve a defense for this vulnerability to disease or, at the very least, to weed this trait out of the population.(({{pubmed>long:10839723}}))   * **[[home:pathogenesis:evolution|evolutionary analysis]] suggests cardiovascular disease is not caused by lifestyle or genetic factors** – Atherosclerosis has been identified in Egyptian mummies.(({{pubmed>long:10495769}})) This suggests that whatever factors cause atherosclerosis has existed for at least several millenia. If it were true that a factor such as a harmful allele for a gene or the consumption of fatty foods caused atherosclerosis, the human species would have had several millenia to evolve a defense for this vulnerability to disease or, at the very least, to weed this trait out of the population.(({{pubmed>long:10839723}}))
   * **prenatal exposure to influenza and cardiovascular disease** – Prenatal exposure to the 1918 influenza pandemic (Influenza A, H1N1 subtype) is associated with >/=20% excess cardiovascular disease at 60 to 82 years of age, relative to cohorts born without exposure to the influenza epidemic, either prenatally or postnatally. These findings suggest novel roles for maternal infections in the fetal programming of cardiovascular risk factors that are independent of maternal malnutrition.(({{pubmed>long:20198106}}))   * **prenatal exposure to influenza and cardiovascular disease** – Prenatal exposure to the 1918 influenza pandemic (Influenza A, H1N1 subtype) is associated with >/=20% excess cardiovascular disease at 60 to 82 years of age, relative to cohorts born without exposure to the influenza epidemic, either prenatally or postnatally. These findings suggest novel roles for maternal infections in the fetal programming of cardiovascular risk factors that are independent of maternal malnutrition.(({{pubmed>long:20198106}}))
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 +[[https://www.amjmedsci.org/article/S0002-9629(15)30809-0/fulltext|Infections May be Causal in the Pathogenesis of Atherosclerosis]]
  
 ==== A causal relationship between periodontal disease and cardiovascular disease? ==== ==== A causal relationship between periodontal disease and cardiovascular disease? ====
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 Hyperuricaemia was associated with an unfavourable cardiovascular risk profile in HF patients. Treatment with low doses of allopurinol did not improve the prognosis of HF patients.  (({{pubmed>long:    31119751}})) Hyperuricaemia was associated with an unfavourable cardiovascular risk profile in HF patients. Treatment with low doses of allopurinol did not improve the prognosis of HF patients.  (({{pubmed>long:    31119751}}))
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 +Increased risks per 10 000 person-years found in estrogen plus progestin therapy for 16608 healthy postmenopausal women (studied over 5 years) were 7 more CHD events, 8 more strokes, 8 more PEs, and 8 more invasive breast cancers. (({{pubmed>long:12117397}}))
  
   * [[http://www.medicalnewstoday.com/articles/91894.php|Olmetec Is First Angiotensin Receptor Blocker (ARB) To Suggest Atherosclerosis Regression (in Hypertensives With Cardiovascular Risk)]]   * [[http://www.medicalnewstoday.com/articles/91894.php|Olmetec Is First Angiotensin Receptor Blocker (ARB) To Suggest Atherosclerosis Regression (in Hypertensives With Cardiovascular Risk)]]
home/diseases/cardiovascular.txt · Last modified: 09.14.2022 by 127.0.0.1
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