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home:diseases:kidney_disease [02.17.2019] – [Kidney disease] sallieqhome:diseases:kidney_disease [05.08.2019] – [Patients experiences] sallieq
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-Am J Kidney Dis. 2009 Apr;53(4):711-4. Epub 2009 Jan 29. +Calcium-alkali syndrome due to vitamin D administration and magnesium oxide administration.(({{pubmed>long:19185403}}))
-Calcium-alkali syndrome due to vitamin D administration and magnesium oxide administration. +
-Hanada S, Iwamoto M, Kobayashi N, Ando R, Sasaki S. +
-Department of Nephrology, Musashino Red Cross Hospital, Tokyo Medical and Dental University, Tokyo, Japan. s-hanada@umin.ac.jp   (({{pubmed>long:19185403}}))+
  
  
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 Got Calcium? Welcome to the Calcium-Alkali Syndrome. Got Calcium? Welcome to the Calcium-Alkali Syndrome.
-Patel AM, Goldfarb S +
-J Am Soc Nephrol Apr 2010; +
-Download citation +
-Affiliation +
-Renal-Electrolyte and Hypertension Division, Department of Medicine, University of Pennsylvania School of Medicine, Philadelphia, Pennsylvania.+
 Abstract Abstract
 We recommend changing the name of the milk-alkali syndrome to the calcium-alkali syndrome, because the new terminology better reflects the shifting epidemiology and understanding of this disorder. The calcium-alkali syndrome is now the third most common cause of hospital admission for hypercalcemia, and those at greatest risk are postmenopausal or pregnant women.  We recommend changing the name of the milk-alkali syndrome to the calcium-alkali syndrome, because the new terminology better reflects the shifting epidemiology and understanding of this disorder. The calcium-alkali syndrome is now the third most common cause of hospital admission for hypercalcemia, and those at greatest risk are postmenopausal or pregnant women. 
  
-The incidence of the calcium-alkali syndrome is growing in large part as a result of the widespread use of over-the-counter calcium and vitamin D supplements.Advertising for treatment or prevention of osteoporosis has long encouraged this use. Intricate mechanisms mediating the calcium-alkali syndrome depend on interplay among intestine, kidney, and bone. New insights regarding its pathogenesis focus on the key role of calcium-sensing receptors and TRPV5 channels in the modulation of renal calcium excretion. Restoring extracellular blood volume, increasing GFR and calcium excretion, and discontinuing calcium supplementation provide best treatment.   (({{pubmed>long:20413609}}))+The incidence of the calcium-alkali syndrome is growing in large part as a result of the widespread use of over-the-counter calcium and vitamin D supplements. Advertising for treatment or prevention of osteoporosis has long encouraged this use.  
 + 
 +Intricate mechanisms mediating the calcium-alkali syndrome depend on interplay among intestine, kidney, and bone. New insights regarding its pathogenesis focus on the key role of calcium-sensing receptors and TRPV5 channels in the modulation of renal calcium excretion.  
 + 
 +Restoring extracellular blood volume, increasing GFR and calcium excretion, and discontinuing calcium supplementation provide best treatment.   (({{pubmed>long:20413609}}))
  
  
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 //**Claudia**, MarshallProtocol.com//</blockquote> //**Claudia**, MarshallProtocol.com//</blockquote>
  
 +==== Patient's chart ====
 +
 +//with notes re eating meat before a test//
 +
 +
 +Creatinine, at 130 umole/L, was above normal range from the start. It took a few months to reach around 150 umole/L which seemed to become the basic level for the the next 11 years with peaks above that level being associated with other factors. The two anomalous peaks above 200umole/L were associated with having meat meals too close to the time of the test. The creatine and creatinine content in vertebrate muscle, or creatine supplements, can be absorbed to add to the body's potential to generate serum creatinine (70Effect of a Cooked Meat Meal on Serum Creatinine and Estimated Glomerular Filtration Rate in Diabetes-Related Kidney Disease | Diabetes Care). After the early 200+ peak, from a sample taken after a light chicken lunch, meat was avoided for a day or two before sampling for subsequent tests. 
  
 +The other exception was the second last one, for which meat was eaten the night before sampling.    Apart from the aberrations arising from meat eating, eGFR values remained in the 30 to 40 range throughout the 11 year period. Thus the MP treatment, while inducing an initial fall in eGFR, has subsequently allowed the kidney function to remain stable. 
  
 +{{:home:tests:kidney_tests..gif?700|}}
  
  
-{{tag>disease kidneys arrange}}+{{tag>disease kidneys }}
  
  
home/diseases/kidney_disease.txt · Last modified: 09.14.2022 by 127.0.0.1
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