This shows you the differences between two versions of the page.
Both sides previous revisionPrevious revisionNext revision | Previous revisionNext revisionBoth sides next revision | ||
home:pathogenesis:th1spectrum [02.09.2019] – [A single diagnosis for chronic inflammatory disease] sallieq | home:pathogenesis:th1spectrum [02.10.2019] – sallieq | ||
---|---|---|---|
Line 441: | Line 441: | ||
A recent survey of Marshall Protocol patients, discussed in [[home: | A recent survey of Marshall Protocol patients, discussed in [[home: | ||
+ | |||
+ | |||
+ | ==== Metabolic syndrome ==== | ||
+ | |||
+ | Impact of 25-hydroxyvitamin D, free and bioavailable fractions of vitamin D, and vitamin D binding protein levels on metabolic syndrome components | ||
+ | |||
+ | |||
+ | ===== Intestinal barrier ===== | ||
+ | |||
+ | Zonulin and its regulation of intestinal barrier function: the biological door to inflammation, | ||
+ | |||
+ | The two major triggers of zonulin release that have been described so far are bacteria and gliadin. It is well described that many enteric pathogens are able to produce enterotoxins that affect the intestinal tight junction of the host. | ||
===== The challenge of diagnosing diseases caused by bacteria ===== | ===== The challenge of diagnosing diseases caused by bacteria ===== | ||
Line 493: | Line 505: | ||
A corollary of this principle is that attempts to compare one patient' | A corollary of this principle is that attempts to compare one patient' | ||
- | |||
- | |||
- | |||
- | ==== Metabolic syndrome ==== | ||
- | |||
- | Impact of 25-hydroxyvitamin D, free and bioavailable fractions of vitamin D, and vitamin D binding protein levels on metabolic syndrome components | ||
- | |||
- | | ||
- | |||
- | | ||
- | |||
- | | ||
- | |||
- | |||
- | |||
- | ===== Intestinal barrier ===== | ||
- | |||
- | Zonulin and its regulation of intestinal barrier function: the biological door to inflammation, | ||
- | |||
- | The two major triggers of zonulin release that have been described so far are bacteria and gliadin. It is well described that many enteric pathogens are able to produce enterotoxins that affect the intestinal tight junction of the host. | ||
- | |||
Line 524: | Line 515: | ||
===== Notes and comments ===== | ===== Notes and comments ===== | ||
+ | |||
+ | |||
+ | | ||
--- //Sallie Q 11.28.2016// | --- //Sallie Q 11.28.2016// |