Table of Contents

Acute respiratory infections

While it is certainly possible to contract an acute respiratory infection while on the Marshall ProtocolA curative medical treatment for chronic inflammatory disease. Based on the Marshall Pathogenesis., many symptoms of immunopathologyA temporary increase in disease symptoms experienced by Marshall Protocol patients that results from the release of cytokines and endotoxins as disease-causing bacteria are killed. mimic those of an acute respiratory infection. Adjusting one's antibiotics or olmesartan (Benicar)Medication taken regularly by patients on the Marshall Protocol for its ability to activate the Vitamin D Receptor. can sometimes help a patient distinguish between the two. The MP tends to make no difference to the course of a common cold. Patients with acute respiratory infections can manage their infections with antiviral agents as well as medicines which palliate symptoms.

“It seems COVID-19 is probably not Pneumonia at all, the microbe attacks the HEME of red blood cells, destroying their ability to absorb oxygen and carbon dioxide so that gently applied supplementary pure oxygen will be an important part of treatment.”

vide: importance of HEME at the 12 minute mark

Distinguishing between acute infection and immunopathology

While it is certainly possible to contract an acute infection while on the MP, many symptoms of immunopathology mimic those of an acute infection. These symptoms include a runny nose or stuffy nose, sore throat, fever, headache, cough, malaise, or vague feeling of illness, muscle aches, loss of appetite, and ear pain. Generally speaking, symptoms due to immunopathology are more variable and tend not to last while a cold, flu, bronchitis, etc. can generally linger a week or more. Whenever the cause of symptoms is in doubt, try one or both of the following:

If symptoms improve in either or both scenarios, it is an immune system reaction, not an acute infection.

Patients who remain unsure if they have an acute infection should consult their physician. A physician can diagnose most acute infections after a history and physical exam. In some cases, further tests may be ordered. For example, the back of the throat may be swabbed if strep throat is suspected. This swab test, called a throat culture, can check for the bacteria that cause this infection. A blood test called an antibody titer may be done if certain infections are suspected. A chest X-ray may be ordered to make sure pneumonia, a more serious infection deep in the lungs, is not present. Other tests may be ordered in certain cases.

Types of acute respiratory infections

Viruses account for most upper respiratory infections, but physicians must be alert to signs of bacterial primary infection or superinfection, which may require targeted therapy.

Managing an acute respiratory infection while on the Marshall Protocol

Certain treatments for acute infections can predispose to chronic disease

The MP tends to make no difference to the course of a common cold. The Vitamin D ReceptorA nuclear receptor located throughout the body that plays a key role in the innate immune response., which is activated by the Marshall Protocol, transcribes at least several genes, which are important for fighting viruses.

If anything, the Marshall Protocol will allow the immune system a better chance to fight the virus.

Trevor Marshall, PhD

Medications

Other measures

Patients who are at risk for symptoms of acute infection should take care of themselves by getting adequate rest, fluids and nutrition. This is especially important for patients who already have compromised respiratory function. It is okay to palliate symptoms with medications as necessary.

What is a virus ?

from https://rationalwiki.org/wiki/Virus

There is some debate as to whether viruses are living organisms or not.[7][8] While they do contain genetic material (i.e. the DNA or RNA), and evolve, like any other living organism, they also contain no cell structures (things like a cytoplasm, cell wall, nucleus, et cetera), lack senses or a metabolism, and cannot reproduce independently.[9] The existence of giant viruses that can be “attacked” in the same way as a cell by smaller viruses, has also led to some controversy regarding the definition of life, and whether viruses fit it.

interestingly wikipedia says

The origins of viruses in the evolutionary history of life are unclear: some may have evolved from plasmids—pieces of DNA that can move between cells—while others may have evolved from bacteria. In evolution, viruses are an important means of horizontal gene transferAny process in which a bacterium inserts genetic material into the genomes of other pathogens or into the genome of its host., which increases genetic diversity in a way analogous to sexual reproduction.[7] Viruses are considered by some biologists to be a life form, because they carry genetic material, reproduce, and evolve through natural selection, although they lack key characteristics (such as cell structure) that are generally considered necessary to count as life. Because they possess some but not all such qualities, viruses have been described as “organisms at the edge of life”

Are viruses alive 1)

Research

Our findings provide direct evidence that TFH play a critical role in vaccine-induced immunity in humans and suggest a novel strategy for promoting such cells by use of intranasal vaccines against respiratory infections. 2)

Members of the NOD-like receptor (NLR) family of pathogen recognition receptors have important roles in orchestrating this response (to invading pathogens) 3)

Read more

Notes and comments

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broken link

1)
Koonin EV, Starokadomskyy P. Are viruses alive? The replicator paradigm sheds decisive light on an old but misguided question. Stud Hist Philos Biol Biomed Sci. 2016 Oct;59:125-34. doi: 10.1016/j.shpsc.2016.02.016. Epub 2016 Mar 7.
[PMID: 26965225] [PMCID: 5406846] [DOI: 10.1016/j.shpsc.2016.02.016]
2)
Aljurayyan A, Puksuriwong S, Ahmed M, Sharma R, Krishnan M, Sood S, Davies K, Rajashekar D, Leong S, McNamara PS, Gordon S, Zhang Q. Activation and Induction of Antigen-Specific T Follicular Helper Cells Play a Critical Role in Live-Attenuated Influenza Vaccine-Induced Human Mucosal Anti-influenza Antibody Response. J Virol. 2018 May 14;92(11):e00114-18. doi: 10.1128/JVI.00114-18. Print 2018 Jun 1.
[PMID: 29563292] [PMCID: 5952133] [DOI: 10.1128/JVI.00114-18]
3)
Lupfer CR, Stokes KL, Kuriakose T, Kanneganti T. Deficiency of the NOD-Like Receptor NLRC5 Results in Decreased CD8(+) T Cell Function and Impaired Viral Clearance. J Virol. 2017 Aug 10;91(17):e00377-17. doi: 10.1128/JVI.00377-17. Print 2017 Sep 1.
[PMID: 28615208] [PMCID: 5553185] [DOI: 10.1128/JVI.00377-17]