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home:alternate:environment [02.15.2019] – [Toxic chemicals] sallieq | home:alternate:environment [09.14.2022] (current) – external edit 127.0.0.1 | ||
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- | Chronic diseases have been around for at least thousands of years, suggesting that a number of the proposed environmental causes for disease, like smoking, man-made toxins and junk food, are not the exclusive causes. Manifestations of both arteriosclerosis(({{pubmed> | + | Chronic diseases have been around for at least thousands of years, suggesting that a number of the proposed environmental causes for disease, like smoking, man-made toxins and junk food, are not the exclusive causes. Manifestations of both arteriosclerosis(({{pmid> |
===== Modern suppression of immune system ===== | ===== Modern suppression of immune system ===== | ||
- | Besides 20th century medical interventions in chronic disease, which overwhelmingly consist of immune suppressive drugs which reduce symptoms in individuals, | + | Besides 20th century medical interventions in chronic disease, which overwhelmingly consist of immune suppressive drugs which reduce symptoms in individuals, |
Attempts to measure and deal with these problems are addressed in a whole new group of articles placed within [[home: | Attempts to measure and deal with these problems are addressed in a whole new group of articles placed within [[home: | ||
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</ | </ | ||
- | But, a 2010 observational study found that increasing fruit and vegetable intake had a marginal impact on risk of cancer.(({{pubmed> | + | But, a 2010 observational study found that increasing fruit and vegetable intake had a marginal impact on risk of cancer.(({{pmid> |
Further, even some of the most ambitious obesity intervention programs, which have gone to great lengths to increase rates of exercise and improve eating habits of a population, have been failures. | Further, even some of the most ambitious obesity intervention programs, which have gone to great lengths to increase rates of exercise and improve eating habits of a population, have been failures. | ||
- | One 1999 $200,000 NIH-funded intervention, | + | One 1999 $200,000 NIH-funded intervention, |
Studies such as these suggest that the official dietary advice given over the last decades do nothing to curb the increase in obesity. But just as the lack of effect of using a 10 day course of penicillin for the treatment of an L-form bacteria infection is not evidence of antibiotics not being helpful in the treatment of L-form bacteria, neither is one specific life style intervention evidence of lifestyle measures not having effect. | Studies such as these suggest that the official dietary advice given over the last decades do nothing to curb the increase in obesity. But just as the lack of effect of using a 10 day course of penicillin for the treatment of an L-form bacteria infection is not evidence of antibiotics not being helpful in the treatment of L-form bacteria, neither is one specific life style intervention evidence of lifestyle measures not having effect. | ||
- | Other interventions, | + | Other interventions, |
Attrition was high in most of these studies, as they usually are in any kind of long term diet studies where major diet components are changed. Therefore these studies are just as much a test of the patients’ motivation and the skills of the health professional giving them advice as a pure diet test. | Attrition was high in most of these studies, as they usually are in any kind of long term diet studies where major diet components are changed. Therefore these studies are just as much a test of the patients’ motivation and the skills of the health professional giving them advice as a pure diet test. | ||
- | The mechanisms behind the effects of a low carbohydrate, | + | The mechanisms behind the effects of a low carbohydrate, |
- | There could however be other mechanisms at play for the effects of a low-carbohydrate diet, such as changes in the body’s (gut) microbiota. (({{pubmed> | + | There could however be other mechanisms at play for the effects of a low-carbohydrate diet, such as changes in the body’s (gut) microbiota. (({{pmid> |
- | In another study, the relative level of the bacterium Selenomonas noxia in the oral cavity was able to predict obesity with a larger than 98 per cent certainty.(({{pubmed> | + | In another study, the relative level of the bacterium Selenomonas noxia in the oral cavity was able to predict obesity with a larger than 98 per cent certainty.(({{pmid> |
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- | In contrast to infectious agents, little evidence implicates typical doses of dietary chemicals as primary causes of human cancer, probably because humans have evolved effective flexible enzymatic systems for degrading potentially carcinogenic chemicals.(({{pubmed> | + | In contrast to infectious agents, little evidence implicates typical doses of dietary chemicals as primary causes of human cancer, probably because humans have evolved effective flexible enzymatic systems for degrading potentially carcinogenic chemicals.(({{pmid> |
{{tag> | {{tag> | ||
+ | < | ||
===== Notes and comments ===== | ===== Notes and comments ===== | ||
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Gut. 2010 Jan; | Gut. 2010 Jan; | ||
- | Gut microbes define liver cancer risk in mice exposed to chemical and viral transgenic hepatocarcinogens.(({{pubmed> | + | Gut microbes define liver cancer risk in mice exposed to chemical and viral transgenic hepatocarcinogens.(({{pmid> |
Fox JG, Feng Y, Theve EJ, Raczynski AR, Fiala JL, Doernte AL, Williams M, McFaline JL, Essigmann JM, Schauer DB, Tannenbaum SR, Dedon PC, Weinman SA, Lemon SM, Fry RC, Rogers AB. | Fox JG, Feng Y, Theve EJ, Raczynski AR, Fiala JL, Doernte AL, Williams M, McFaline JL, Essigmann JM, Schauer DB, Tannenbaum SR, Dedon PC, Weinman SA, Lemon SM, Fry RC, Rogers AB. | ||
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PMID: 19850960</ | PMID: 19850960</ | ||
- | ===== References ===== | + | ===== References =====</ |