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home:diseases:anxiety [01.26.2020] – [Evidence of infectious cause] sallieq | home:diseases:anxiety [09.14.2022] (current) – external edit 127.0.0.1 | ||
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autism, ADHD, depression, severe anxiety, chronic fatigue syndrome (CFS) | autism, ADHD, depression, severe anxiety, chronic fatigue syndrome (CFS) | ||
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- | //**L.E. Goehler** et al.//(({{pubmed> | + | //**L.E. Goehler** et al.//(({{pmid> |
- | Further, Neufeld //et al.// showed that germ-free mice exhibited reduced anxiety-like behavior as well as significant neurochemical changes in the brain compared to specific-pathogen-free mice. (({{pubmed> | + | Further, Neufeld //et al.// showed that germ-free mice exhibited reduced anxiety-like behavior as well as significant neurochemical changes in the brain compared to specific-pathogen-free mice. (({{pmid> |
- | The frequency with which patients of certain Th1 diseases also experience anxiety(({{pubmed> | + | The frequency with which patients of certain Th1 diseases also experience anxiety(({{pmid> |
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+ | Findings suggest that a low dose of THC produces subjective stress-relieving effects in line with those commonly reported among cannabis users, but that higher doses may non-specifically increase negative mood. (({{pmid> | ||
+ | Baseline symptoms of depression (but not anxiety or stress) appeared to be exacerbated across time/ | ||
+ | Perhaps most well-studied is the role the CeA plays in unconditioned and conditioned fear generation (Ciocchi et al., 2010, Li et al., 2013, Tye et al., 2011), fear extinction, and conditioned inhibition (Amano et al., 2010), as well as conditioned orienting responses to emotionally salient stimuli (El-Amamy and Holland, 2007, Groshek et al., 2005). | ||
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+ | the mechanisms by which stress increases amygdala-dmPFC synaptic strength and generates anxiety-like behaviors are not well understood. Here, we show that the mouse basolateral amygdala (BLA)-prelimbic prefrontal cortex (plPFC) circuit is engaged by stress and activation of this pathway in anxiogenic. [[https:// | ||
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===== Notes and comments ===== | ===== Notes and comments ===== | ||
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Bacteria’s presence or absence affected how the mice used certain brain chemicals and genes involved in brain development. Taken together, the results indicate that intestinal bacteria somehow shape the brain and make mice more anxious — or cautious, depending how you look at it, Pettersson says. | Bacteria’s presence or absence affected how the mice used certain brain chemicals and genes involved in brain development. Taken together, the results indicate that intestinal bacteria somehow shape the brain and make mice more anxious — or cautious, depending how you look at it, Pettersson says. | ||
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- | ===== References ===== | + | ===== References =====</ |