This shows you the differences between two versions of the page.
Both sides previous revisionPrevious revisionNext revision | Previous revisionNext revisionBoth sides next revision | ||
home:diseases:cancer [04.07.2019] – [see also M P Study Site Discussion] sallieq | home:diseases:cancer [06.29.2019] – sallieq | ||
---|---|---|---|
Line 23: | Line 23: | ||
* **liver cancer** – It is "well established" | * **liver cancer** – It is "well established" | ||
* **lung cancer** – According to a 2011 meta-analysis, | * **lung cancer** – According to a 2011 meta-analysis, | ||
- | * **prostate cancer** – In a 2005 culture-based study, Cohen found that // | + | * **prostate cancer** – In a 2005 culture-based study, Cohen found that // |
+ | * **Table of cancers** with associated agents. [[https:// | ||
Line 75: | Line 76: | ||
- | ==== Are There Concerns About Supplementing with Vitamin | + | ==== Are There Concerns About V.D Supplement |
There is a common misconception that vitamin D supplementation is safe at any reasonable level, or that if some is good, more may be better. | There is a common misconception that vitamin D supplementation is safe at any reasonable level, or that if some is good, more may be better. | ||
Line 87: | Line 88: | ||
Vitamin D receptor-mediated skewed differentiation of macrophages initiates myelofibrosis and subsequent osteosclerosis. (({{pubmed> | Vitamin D receptor-mediated skewed differentiation of macrophages initiates myelofibrosis and subsequent osteosclerosis. (({{pubmed> | ||
+ | |||
+ | 2014 a Cochrane meta-analysis that included 18 randomized clinical trials comparing vitamin D administration versus no intervention in healthy population found no difference regarding cancer incidence. | ||
====1, | ====1, | ||
Line 97: | Line 100: | ||
- | Women with hereditary | + | Women with hereditary cancer predispositions should |
Glioblastomas and malignant gliomas are the most common primary malignant brain tumors, with an annual incidence of 5.26 per 100,000 population. These tumors are typically associated with a dismal prognosis and poor quality of life. Only radiation exposure and certain genetic syndromes are well-defined risk factors for malignant glioma. | Glioblastomas and malignant gliomas are the most common primary malignant brain tumors, with an annual incidence of 5.26 per 100,000 population. These tumors are typically associated with a dismal prognosis and poor quality of life. Only radiation exposure and certain genetic syndromes are well-defined risk factors for malignant glioma. | ||
+ | |||
+ | This compound has antiproliferative and antimigratory effects in squamous cell carcinoma, glioblastoma, | ||
Several researchers have highlighted a possible link between glioma and human cytomegalovirus (HCMV). | Several researchers have highlighted a possible link between glioma and human cytomegalovirus (HCMV). | ||
Line 110: | Line 115: | ||
+ | ===== Microbial involvement ===== | ||
- | ===== Single species vs. communities of microbes | + | ==== Single species vs. communities of microbes ==== |
Alicia H. Chang and Julie Parsonnet of Stanford University writes that a transmissible cause of cancer was suspected as early as the 16th century. It was not until the late 20th century definitively identified a bacterial cause of at least some types of cancer to most researchers' | Alicia H. Chang and Julie Parsonnet of Stanford University writes that a transmissible cause of cancer was suspected as early as the 16th century. It was not until the late 20th century definitively identified a bacterial cause of at least some types of cancer to most researchers' | ||
Line 122: | Line 128: | ||
- | ===== Slow-growing microbes elicit inflammation | + | ==== Slow-growing microbes elicit inflammation ==== |
< | < | ||
Line 137: | Line 143: | ||
- | ===== Inflammation induces cancer | + | ==== Inflammation induces cancer ==== |
According to Chang, | According to Chang, | ||
Line 149: | Line 155: | ||
- | ===== Microbial interaction and disease | + | ==== Microbial interaction and disease ==== |
According to some sporadic reports, cancer regresses following spontaneous bacterial infection.(({{pubmed> | According to some sporadic reports, cancer regresses following spontaneous bacterial infection.(({{pubmed> | ||
Line 163: | Line 169: | ||
- | ===== Metastatic communities parallel biofilm | + | ==== Metastatic communities parallel biofilm ==== |
Some have suggested provocatively that communities of metastatic cancerous cells closely resemble communities of microbes known as biofilm. | Some have suggested provocatively that communities of metastatic cancerous cells closely resemble communities of microbes known as biofilm. | ||
Line 190: | Line 196: | ||
- | + | ==== Tobacco ==== | |
- | ====Tobacco==== | + | |
{{section>: | {{section>: | ||
+ | |||
+ | |||
+ | ==== Stress derived from interaction of particular personalities ==== | ||
+ | |||
+ | in particular [[https:// | ||
+ | |||
+ | from https:// | ||
+ | |||
+ | < | ||
+ | |||
+ | In an extreme case, there may be someone who is literally toxic, even socio-pathic. “Learn to value yourself, Learn to say No!” and if necessary, Remove yourself from the influence of any such person.</ | ||
Line 256: | Line 272: | ||
//**Gene**, MarshallProtocol.com//</ | //**Gene**, MarshallProtocol.com//</ | ||
- | ====Vitamin D======== | + | ==== Vitamin D==== |
< | < | ||
Line 262: | Line 278: | ||
+ | === Vitamin E === | ||
+ | Vitamin E supplementation and the risk of heart failure in women. | ||
+ | (({{pubmed> | ||
+ | Supplementation with vitamin E did not result in any significant improvements in prognostic or functional indexes of heart failure or in the quality of life of patients with advanced heart failure.(({{pubmed> | ||
+ | |||
+ | Canolol was found to counteract the fibrotic effects of vitamin E + CoQ10 on cardiac fibrosis in the context of a high-fat diet enriched with RSO. This effect occurred through a restoration of cardiac Ag2R-1b mRNA expression and decreased ischemia.(({{pubmed> | ||
Line 363: | Line 385: | ||
===== Recent research ===== | ===== Recent research ===== | ||
- | |||
Paclitaxel Reduces Tumor Growth by Reprogramming Tumor-Associated Macrophages to an M1 Profile in a TLR4-Dependent Manner. | Paclitaxel Reduces Tumor Growth by Reprogramming Tumor-Associated Macrophages to an M1 Profile in a TLR4-Dependent Manner. | ||
- | |||
{{section>: | {{section>: | ||
Line 376: | Line 396: | ||
In this large population-based gastro-oesophageal cancer cohort, we found moderately reduced cancer-specific mortality among ARB users. However, confirmation in further independent epidemiological studies with sufficient staging information is required. | In this large population-based gastro-oesophageal cancer cohort, we found moderately reduced cancer-specific mortality among ARB users. However, confirmation in further independent epidemiological studies with sufficient staging information is required. | ||
- | miR-205 mediates the inhibition of cervical cancer cell proliferation using olmesartan. | + | miR-205 mediates the inhibition of cervical cancer cell proliferation using olmesartan. |
Therapeutic potentials of SCFA receptors [section 7 in | Therapeutic potentials of SCFA receptors [section 7 in | ||
Recent progress in understanding the role of GPR41, GPR43 and GPR109A in health and disease demonstrate that these receptors do not just regulate inflammation and cancer in intestine, but also influence other gastrointestinal functions, allergies, adipogenesis, | Recent progress in understanding the role of GPR41, GPR43 and GPR109A in health and disease demonstrate that these receptors do not just regulate inflammation and cancer in intestine, but also influence other gastrointestinal functions, allergies, adipogenesis, | ||
+ | | ||
+ | |||
+ | | ||
+ | |||
+ | Using metabolomics, | ||
Line 392: | Line 417: | ||
* [[https:// | * [[https:// | ||
* [[https:// | * [[https:// | ||
- | * [[http:// | + | * [[http:// |
* [[http:// | * [[http:// | ||