Home

Differences

This shows you the differences between two versions of the page.

Link to this comparison view

Both sides previous revisionPrevious revision
Next revision
Previous revision
Next revisionBoth sides next revision
home:diseases:cancer [04.07.2019] – [Read more] sallieqhome:diseases:cancer [04.26.2019] – [Are There Concerns About V.D Supplement ?] sallieq
Line 23: Line 23:
   * **liver cancer** – It is "well established" that chronic infections with hepatitis B and C viruses (HBV, HCV) represent major risk factors for HCC.(({{pubmed>long:10710202}}))   * **liver cancer** – It is "well established" that chronic infections with hepatitis B and C viruses (HBV, HCV) represent major risk factors for HCC.(({{pubmed>long:10710202}}))
   * **lung cancer** – According to a 2011 meta-analysis, patients exposed to //C. pneumoniae// infection had a 48% greater risk for lung cancer risk, relative to those not exposed.(({{pubmed>long:21194924}}))   * **lung cancer** – According to a 2011 meta-analysis, patients exposed to //C. pneumoniae// infection had a 48% greater risk for lung cancer risk, relative to those not exposed.(({{pubmed>long:21194924}}))
-  * **prostate cancer** – In a 2005 culture-based study, Cohen found that //Propionibacterium acnes// in 35% of prostate samples of 34 consecutive patients tested.(({{pubmed>long:15879794}})) +  * **prostate cancer** – In a 2005 culture-based study, Cohen found that //Propionibacterium acnes// in 35% of prostate samples of 34 consecutive patients tested.(({{pubmed>long:15879794}})) 
 +  * **Table of cancers** with associated agents. [[https://bmccancer.biomedcentral.com/track/pdf/10.1186/s12885-017-3234-4|Infections and Cancer]]
  
  
Line 87: Line 88:
  
 Vitamin D receptor-mediated skewed differentiation of macrophages initiates myelofibrosis and subsequent osteosclerosis. (({{pubmed>long:30718230}}))  Vitamin D receptor-mediated skewed differentiation of macrophages initiates myelofibrosis and subsequent osteosclerosis. (({{pubmed>long:30718230}})) 
 +
 +2014  a Cochrane meta-analysis that included 18 randomized clinical trials comparing vitamin D administration versus no intervention in healthy population found no difference regarding cancer incidence.  In randomised controlled trials, relative risks for all cause mortality were 0.89 (0.80 to 0.99) for vitamin D3 supplementation and 1.04 (0.97 to 1.11) for vitamin D2 supplementation. The effects observed for vitamin D3 supplementation remained unchanged when grouped by various characteristics. However, for vitamin D2 supplementation, increased risks of mortality were observed in studies with lower intervention doses and shorter average intervention periods.  (({{pubmed>long:    24690623}}))
  
 ====1,25-dihydroxyvitamin D ==== ====1,25-dihydroxyvitamin D ====
Line 256: Line 259:
  
 //**Gene**, MarshallProtocol.com//</blockquote>  //**Gene**, MarshallProtocol.com//</blockquote> 
-====Vitamin D========+==== Vitamin D====
 <mainarticle> [[home:pathogenesis:vitamind:cancer|Vitamin D and cancer]] </article> <mainarticle> [[home:pathogenesis:vitamind:cancer|Vitamin D and cancer]] </article>
  
 {{section>:home:pathogenesis:vitamind:cancer#vitamin_D_and_cancer&noheader&firstseconly}} {{section>:home:pathogenesis:vitamind:cancer#vitamin_D_and_cancer&noheader&firstseconly}}
  
 +
 +=== Vitamin E ===
  
  
 +Vitamin E supplementation and the risk of heart failure in women.
 +(({{pubmed>long:    22438520}}))
  
 +Supplementation with vitamin E did not result in any significant improvements in prognostic or functional indexes of heart failure or in the quality of life of patients with advanced heart failure.(({{pubmed>long:    11157316}}))
  
 +Canolol was found to counteract the fibrotic effects of vitamin E + CoQ10 on cardiac fibrosis in the context of a high-fat diet enriched with RSO. This effect occurred through a restoration of cardiac Ag2R-1b mRNA expression and decreased ischemia.(({{pubmed>long:    29456586}}))
  
  
home/diseases/cancer.txt · Last modified: 09.14.2022 by 127.0.0.1
© 2015, Autoimmunity Research Foundation. All Rights Reserved.