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home:diseases:cardiovascular [12.06.2018] – [Homocysteine] + ref. (uric acid) sallieq | home:diseases:cardiovascular [04.09.2020] – [Other evidence] sallieq | ||
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- | ====== Cardiovascular diseases ====== | + | ====== Cardiovascular diseases ====== |
- | <relatedarticle> [[home: | + | <relatedarticles> [[home: |
Heart disease or cardiovascular diseases is the class of diseases that involve the heart or blood vessels (arteries and veins). While the term technically refers to any disease that affects the cardiovascular system, it is usually used to refer to those related to arteriosclerosis (hardening of the arteries). | Heart disease or cardiovascular diseases is the class of diseases that involve the heart or blood vessels (arteries and veins). While the term technically refers to any disease that affects the cardiovascular system, it is usually used to refer to those related to arteriosclerosis (hardening of the arteries). | ||
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+ | Macrophages are central to atherogenesis because they regulate cholesterol traffic and inflammation in the arterial wall. < | ||
According to the Marshall Pathogenesis, | According to the Marshall Pathogenesis, | ||
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* **Common infections predispose a person to stroke and heart attack** – In a prospective cohort study, a composite measure of //Chlamydia pneumoniae, Helicobacter pylori//, cytomegalovirus, | * **Common infections predispose a person to stroke and heart attack** – In a prospective cohort study, a composite measure of //Chlamydia pneumoniae, Helicobacter pylori//, cytomegalovirus, | ||
* **Common infections and cardiovascular diseases share the same inflammatory markers** – As Costa //et al.// have pointed out, | * **Common infections and cardiovascular diseases share the same inflammatory markers** – As Costa //et al.// have pointed out, | ||
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* **prenatal exposure to influenza and cardiovascular disease** – Prenatal exposure to the 1918 influenza pandemic (Influenza A, H1N1 subtype) is associated with >/=20% excess cardiovascular disease at 60 to 82 years of age, relative to cohorts born without exposure to the influenza epidemic, either prenatally or postnatally. These findings suggest novel roles for maternal infections in the fetal programming of cardiovascular risk factors that are independent of maternal malnutrition.(({{pubmed> | * **prenatal exposure to influenza and cardiovascular disease** – Prenatal exposure to the 1918 influenza pandemic (Influenza A, H1N1 subtype) is associated with >/=20% excess cardiovascular disease at 60 to 82 years of age, relative to cohorts born without exposure to the influenza epidemic, either prenatally or postnatally. These findings suggest novel roles for maternal infections in the fetal programming of cardiovascular risk factors that are independent of maternal malnutrition.(({{pubmed> | ||
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==== A causal relationship between periodontal disease and cardiovascular disease? ==== | ==== A causal relationship between periodontal disease and cardiovascular disease? ==== | ||
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{{section>: | {{section>: | ||
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===== Other treatments ===== | ===== Other treatments ===== | ||
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//**Jen Hicks**, MarshallProtocol.com//</ | //**Jen Hicks**, MarshallProtocol.com//</ | ||
===== More information ===== | ===== More information ===== | ||
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+ | Hyperuricaemia was associated with an unfavourable cardiovascular risk profile in HF patients. Treatment with low doses of allopurinol did not improve the prognosis of HF patients. | ||
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+ | Increased risks per 10 000 person-years found in estrogen plus progestin therapy for 16608 healthy postmenopausal women (studied over 5 years) were 7 more CHD events, 8 more strokes, 8 more PEs, and 8 more invasive breast cancers. (({{pubmed> | ||
* [[http:// | * [[http:// |