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home:diseases:diabetes2 [02.18.2019] – [Evidence of infectious cause] sallieq | home:diseases:diabetes2 [02.19.2019] – [Antimicrobial therapy in insulin resistance/type 2 diabetes] sallieq | ||
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- | Diet and exercise are the traditional lifestyle interventions used to prevent and treat type 2 diabetes. Different components of the diet can affect the course of type 2 diabetes, but a low carbohydrate diet appears to be giving the most significant results. (({{pubmed> | + | Diet and exercise are the traditional lifestyle interventions used to prevent and treat type 2 diabetes. Different components of the diet can affect the course of type 2 diabetes, but a low carbohydrate diet appears to be giving the most significant results. (({{pubmed> |
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+ | Both endurance and resistance exercise yield improvements in many aspects of the metabolic derangements in diabetes type II.(({{pubmed> | ||
Sulfonylureas, | Sulfonylureas, | ||
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The use of oxytetracycline in obese mice reduced insulin resistance(({{pubmed> | The use of oxytetracycline in obese mice reduced insulin resistance(({{pubmed> | ||
- | Diabetes can cause a sugar coating that smothers body's immune defences | ||
- | http:// | ||
- | The researchers looked at the similarities in chemical structure between glucose in blood and body fluids, and two other sugar called mannose and fucose. These sugars are found on the surfaces of bacteria and fungi and act as targets for receptors in our body that have evolved to detect and bind to microbial sugars to then combat the infection. | + | Diabetes can cause a sugar coating that smothers |
- | The research found that high levels of glucose | + | The researchers looked at the similarities in chemical structure between |
- | • It can inhibit the function of immune system receptors called C-type lectins such as MBL (Mannose-binding lectin) which are known to bind to a sugar known as mannose that is present in the structure of infectious fungal bacterial cell walls. Unlike glucose, mannose does not exist in mammals as a free sugar in the blood. | + | |
- | • The loss of MBL function may also predispose | + | The research found that high levels |
- | • A number of C-type lectins tat can be affected by raised | + | < |
- | Warwick Medical School researcher Dr Daniel Mitchell said: | ||
- | "Our findings offer a new perspective on how high glucose can potentially affect immunity and thus exert a negative impact on health. It also helps to emphasize the importance of good diet on preventing or controlling diseases such as diabetes. We will build on these ideas in order to consolidate the disease model and to investigate new routes to treatment and prevention." | ||
+ | Rubella virus-induced diabetes in the hamster. | ||
+ | < | ||
- | 60. Rayfield EJ, Kelly KJ, Yoon JW. Rubella virus-in- | ||
- | duced diabetes in the hamster. Diabetes. | ||
- | 1986; | ||
- | 61. Menser MA, Forrest JM, Bransby RD. Rubella in- | ||
- | fection and diabetes mellitus. Lancet. 1978; | ||
- | 62. Ramsingh AI, Chapman N, Tracy S. | ||
- | Coxsackieviruses and diabetes. Bioessays. | ||
- | 1997; | ||
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