- For Patients
- Introduction to the Marshall Protocol
- Length of the MP
- Immunopathology
- Managing immunopathology
- Food and drink
- Light restriction
- Vitamin D metabolite calculator - get feedback on vitamin D results
- Therapeutic probe - best method for determining MP suitability
- Starting the Marshall Protocol
- Resources for patients
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- Patient interviews on Bacteriality
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- Science behind immunopathology
- Science behind olmesartan (Benicar)
- Safety of olmesartan
- Resources for physicians
- Vitamin D metabolite calculator - get feedback on vitamin D results
- Therapeutic probe - best method for determining MP suitability
- Physicians' concerns about the MP
- Notice for emergency medical personnel
- The Pathogenesis
- Publications and presentations
- Science behind Marshall Pathogenesis
- Microbes in the human body
- Successive infection and variability in disease
- Autoimmune theory of disease
- Science behind vitamin D
- Metabolism of vitamin D and the VDR
- Th1 Spectrum Disorder - how chronic inflammatory diseases are related
- Transmission of bacteria and onset of chronic disease
- Evolutionary perspective on disease
- Incidence and prevalence of disease
- Evidence that chronic disease is caused by pathogens
- Diseases
- Foundation
- Related Sites
Differences
This shows you the differences between two versions of the page.
| Both sides previous revisionPrevious revisionNext revision | Previous revisionNext revisionBoth sides next revision | ||
| home:diseases:hlh [07.26.2010] – inge | home:diseases:hlh [07.26.2010] – inge | ||
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| ==== Possible mechanism for hemophagocytosis ==== | ==== Possible mechanism for hemophagocytosis ==== | ||
| - | The actual hemophagocytosis may be viewed as a result of overstimulated macrophages. While blood cells may harbour infectious agents and therefore the infected individual could benefit from the macrophages attacking the blood cells, there is also the possibility that hemophagocytosis benefits the infectious agent. Bacteria in hemophagocytic macrophages could access nutrients from the ingested cells, or hemophagocytic macrophages could reduce bacteria-killing activity by killing the very cells that could attack them. Yet, other macrophages could still be sufficiently activated to generate additional hemophagocytic macrophages. (({{pubmed> | + | The actual hemophagocytosis may be viewed as a result of overstimulated macrophages. While blood cells may harbour infectious agents and therefore the infected individual could benefit from the macrophages attacking the blood cells, there is also the possibility that hemophagocytosis benefits the infectious agent. Bacteria in hemophagocytic macrophages could access nutrients from the ingested cells, or hemophagocytic macrophages could reduce bacteria-killing activity by killing the very cells that could attack them. Yet, other macrophages could still be sufficiently activated to generate additional hemophagocytic macrophages. (({{pubmed> |
| ==== Relation to chronic infection ==== | ==== Relation to chronic infection ==== | ||
| Line 28: | Line 28: | ||
| REVISE | REVISE | ||
| - | So far, so good, Inge. Note the syntax for references.(({{pubmed> | + | So far, so good, Inge. Note the syntax for references. |
| ===== References ===== | ===== References ===== | ||
home/diseases/hlh.txt · Last modified: 09.14.2022 by 127.0.0.1
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