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home:diseases [09.30.2011] – [Notes and comments] ingehome:diseases [11.10.2011] – [Notes and comments] paulalbert
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 http://tiny.cc/1lxnw http://tiny.cc/1lxnw
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 +High levels of Epstein–Barr virus in COPD
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 +http://erj.ersjournals.com/content/31/6/1221.long
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 +COPD is related to the EBV Virus, which may potentiate other Viruses/Pathogens:
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 +Thus, chronic EBV infection may lead to impaired interferon-γ production and, in turn, a reduced innate immune response characterised by recurrent viral and bacterial infection.
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 +Also interesting is that people with COPD have  high amounts of the bacteria pseudomonas, plus other bacterias. When one smokes, it is like injecting pathogens into the blood stream, via the lungs.
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 +http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2854762/
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 +Excerpt:
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 +Results
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 +Fifteen different classes of bacteria and a broad range of potentially pathogenic organisms were detected in all cigarette samples. Most notably, we detected Acinetobacter, Bacillus, Burkholderia, Clostridium, Klebsiella, Pseudomonas aeruginosa, and Serratia in ≥ 90% of all cigarette samples. Other pathogenic bacteria detected included Campylobacter, Enterococcus, Proteus, and Staphylococcus. No significant variability in bacterial diversity was observed across the four different cigarette brands.
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 +Conclusions
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 +Previous studies have shown that smoking is associated with colonization by pathogenic bacteria and an increased risk of lung infections. However, this is the first study to show that cigarettes themselves could be the direct source of exposure to a wide array of potentially pathogenic microbes among smokers and other people exposed to secondhand smoke. The overall public health implications of these findings are unclear at this time, and future studies are necessary to determine whether bacteria in cigarettes could play important roles in the development of both infectious and chronic respiratory diseases.
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 +From: LeeDate: 2011-11-01 07:44:01
 +Reply: http://www.marshallprotocol.com/reply.php?topic_id=13718
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 +When I tested positive for EBV the doc said I had the highest titers they had even seen in that clinic ....:shock:   I am a non-smoker DX- Sarcoidosis with COPD and emphysema.  Lee
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 +From: eClaire
 +Date: 2011-11-01 11:07:07
 +Reply: http://www.marshallprotocol.com/reply.php?topic_id=13718
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 +I have COPD and had two different strains of EBV in the late 90s alone. Who knows when I was younger? I doubt I was ever tested.
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 +I was raised by smokers. The COPD came to light when having some sort of cardiogram... the person had trouble seeing my heart because of my damaged lungs. She said my lungs looked like that of someone who was a heavy smoker or had had very bad asthma throughout life. This much I know: when my immune system would go on a tear related to tobacco smoke, VOCs in paint, allergy to cats, etc., my lungs would produce an oatmeal like substance and sometimes I'd stay awake steaming my lungs because it felt like I had to focus on breathing to get oxygen. Even my allergist was puzzled by what my lungs produced when I was in this state.
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 +<blockquote>Anaerobe. 2011 Aug;17(4):191-5. Epub 2011 Mar 3.Molecular methods to describe the spectrum and dynamics of the vaginal microbiota.
 +Fredricks DN.
 +Source
 +Fred Hutchinson Cancer Research Center, Vaccine and Infectious Disease Division, Seattle, WA 98109-1024, USA. dfredric@fhcrc.org
 +Abstract
 +The human vagina hosts a collection of microbes that is distinct from other human surfaces and mucosal sites, with reduced microbial diversity that is likely driven by the acidic environment. The microbial ecosystem of the vagina is dominated by lactobacilli in women without bacterial vaginosis (BV), and is characterize by increased species richness, diversity, and evenness in women with BV. The use of molecular, cultivation-independent methods to describe the bacterial biota of the human vagina has revealed many novel putative anaerobes in women with BV, and has demonstrated the almost ubiquitous nature of Lactobacillus iners which is found in most women regardless of BV status. A variety of molecular tools are being employed to study the vaginal microbiota, and each approach has distinct advantages and disadvantages that are reviewed. Longitudinal studies have demonstrated that the vaginal microbiota can be highly dynamic, with dramatic shifts in bacterial composition and concentrations in response to numerous endogenous and exogenous factors.
 +Copyright © 2011 Elsevier Ltd. All rights reserved.
 +PMID: 21376827 
  
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