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home:diseases [11.10.2011] – [Notes and comments] paulalberthome:diseases [05.29.2020] – sallieq
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 Some diseases are represented by more patients trying the therapy than others. As more patients join the MP cohort, the Autoimmunity Research Foundation will gather more data about the efficacy of the treatment with respect to individual diseases. In the meantime, the MP may be an appropriate treatment option for some of the diseases listed below.  Some diseases are represented by more patients trying the therapy than others. As more patients join the MP cohort, the Autoimmunity Research Foundation will gather more data about the efficacy of the treatment with respect to individual diseases. In the meantime, the MP may be an appropriate treatment option for some of the diseases listed below. 
  
-[[home:patients:patient_interviews|Approximately twenty patients]] have shared their stories of recovery on [[http://bacteriality.com/|Bacteriality.com]].+[[home:patients:patient_interviews|Many patients]] have shared their stories of recovery on [[https://www.marshallprotocol.com/|Marshall protocol study site]].
  
 ===== What is a chronic inflammatory disease? ===== ===== What is a chronic inflammatory disease? =====
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 Although patients who become infected with the Th1 pathogens are given a variety of diagnoses, there are often no clear cut distinctions between one disease and the next. Rather, symptoms frequently overlap creating a spectrum of illness in which diseases are more connected to one another than mutually exclusive disease states. Although patients who become infected with the Th1 pathogens are given a variety of diagnoses, there are often no clear cut distinctions between one disease and the next. Rather, symptoms frequently overlap creating a spectrum of illness in which diseases are more connected to one another than mutually exclusive disease states.
 +
 +===== Microbial communication =====
 +
 + Here we show that an EHEC luxS mutant, unable to produce the bacterial autoinducer, still responds to a eukaryotic cell signal to activate expression of its virulence genes. We have identified this signal as the hormone epinephrine and show that β- and α-adrenergic antagonists can block the bacterial response to this hormone.  (({{pubmed>long:12847292}}))
 +
 +The contribution of cell-cell signaling and motility to bacterial biofilm formation.  (({{pubmed>long:22053126}}))
 +  
 +Bacterial Biofilm Control by Perturbation of Bacterial Signaling Processes.(({{pubmed>long:28902153}}))
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 +
 ===== List of diseases ===== ===== List of diseases =====
    
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 ===== Notes and comments ===== ===== Notes and comments =====
-EDIT+
  
 <blockquote> <blockquote>
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 I was raised by smokers. The COPD came to light when having some sort of cardiogram... the person had trouble seeing my heart because of my damaged lungs. She said my lungs looked like that of someone who was a heavy smoker or had had very bad asthma throughout life. This much I know: when my immune system would go on a tear related to tobacco smoke, VOCs in paint, allergy to cats, etc., my lungs would produce an oatmeal like substance and sometimes I'd stay awake steaming my lungs because it felt like I had to focus on breathing to get oxygen. Even my allergist was puzzled by what my lungs produced when I was in this state. I was raised by smokers. The COPD came to light when having some sort of cardiogram... the person had trouble seeing my heart because of my damaged lungs. She said my lungs looked like that of someone who was a heavy smoker or had had very bad asthma throughout life. This much I know: when my immune system would go on a tear related to tobacco smoke, VOCs in paint, allergy to cats, etc., my lungs would produce an oatmeal like substance and sometimes I'd stay awake steaming my lungs because it felt like I had to focus on breathing to get oxygen. Even my allergist was puzzled by what my lungs produced when I was in this state.
 +
 +</blockquote>
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 +<blockquote>Anaerobe. 2011 Aug;17(4):191-5. Epub 2011 Mar 3.Molecular methods to describe the spectrum and dynamics of the vaginal microbiota.
 +Fredricks DN.
 +Source
 +Fred Hutchinson Cancer Research Center, Vaccine and Infectious Disease Division, Seattle, WA 98109-1024, USA. dfredric@fhcrc.org
 +Abstract
 +The human vagina hosts a collection of microbes that is distinct from other human surfaces and mucosal sites, with reduced microbial diversity that is likely driven by the acidic environment. The microbial ecosystem of the vagina is dominated by lactobacilli in women without bacterial vaginosis (BV), and is characterize by increased species richness, diversity, and evenness in women with BV. The use of molecular, cultivation-independent methods to describe the bacterial biota of the human vagina has revealed many novel putative anaerobes in women with BV, and has demonstrated the almost ubiquitous nature of Lactobacillus iners which is found in most women regardless of BV status. A variety of molecular tools are being employed to study the vaginal microbiota, and each approach has distinct advantages and disadvantages that are reviewed. Longitudinal studies have demonstrated that the vaginal microbiota can be highly dynamic, with dramatic shifts in bacterial composition and concentrations in response to numerous endogenous and exogenous factors.
 +Copyright © 2011 Elsevier Ltd. All rights reserved.
 +PMID: 21376827 
  
 </blockquote> </blockquote>
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