This shows you the differences between two versions of the page.
Both sides previous revisionPrevious revisionNext revision | Previous revisionNext revisionBoth sides next revision | ||
home:mp:stages [10.13.2018] – [Read more] sallieq | home:mp:stages [12.14.2018] – [Stage 3: Benicar partially activates the VDR] sallieq | ||
---|---|---|---|
Line 9: | Line 9: | ||
- | =====Stage 1: Healthy Vitamin D Receptor (VDR), low pathogen load ===== | + | ===== Prior to immune dysfunction |
In a healthy human, the body can carefully regulate activity of the VDR, which it does by controlling the concentrations of two key forms of vitamin D: 25-D and 1,25-D. Both molecules are attracted to the Receptor and bind to it for hours at a time, after which they drop off. The essential difference between the key molecules is that when 1,25-D docks to the VDR, it turns the Receptor on; when 25-D docks to the VDR, it is turned off. When it becomes necessary to turn on the VDR, the body increases production of 1,25-D. Conversely, when an active VDR is no longer necessary, the body converts 1,25-D into 25-D. In healthy humans, this control mechanism works well. For example, in the event a pathogen is noticed, the body increases production of 1,25-D, which in turn increases activity of the VDR. | In a healthy human, the body can carefully regulate activity of the VDR, which it does by controlling the concentrations of two key forms of vitamin D: 25-D and 1,25-D. Both molecules are attracted to the Receptor and bind to it for hours at a time, after which they drop off. The essential difference between the key molecules is that when 1,25-D docks to the VDR, it turns the Receptor on; when 25-D docks to the VDR, it is turned off. When it becomes necessary to turn on the VDR, the body increases production of 1,25-D. Conversely, when an active VDR is no longer necessary, the body converts 1,25-D into 25-D. In healthy humans, this control mechanism works well. For example, in the event a pathogen is noticed, the body increases production of 1,25-D, which in turn increases activity of the VDR. | ||
- | =====Stage 2: Unhealthy VDR, pathogens downregulate | + | ===== Process of immune |
Multiple pathogens including bacteria such as " | Multiple pathogens including bacteria such as " | ||
Line 24: | Line 24: | ||
- | =====Stage 3: Benicar | + | ===== Olmesartan Medoxomil |
The Marshall Protocol (MP) is designed to interrupt the inflammatory disease process. The most important medication Marshall Protocol patients take is not the low doses of antibiotics, | The Marshall Protocol (MP) is designed to interrupt the inflammatory disease process. The most important medication Marshall Protocol patients take is not the low doses of antibiotics, |