Differences

This shows you the differences between two versions of the page.

--- home:othertreatments:antibacterials:highdose [06.29.2010] – paulalbert
+++ home:othertreatments:antibacterials:highdose [01.03.2012] – external edit 127.0.0.1
@@ Line 7: / Line 7: @@
 Antibiotic protocols and treatments other than the Marshall Protocol have been widely prescribed for certain Th1 diseases including rheumatoid arthritis and Lyme disease. However, the evidence for these treatments' efficacy is limited to short-term improvement and patients tend to relapse.
-Although the Th1 diseases are caused by bacterial pathogens, alternatives to the Marshall Protocol are ineffective for at least several reasons. For one, antibiotics given in high enough doses interfere with immune activity. With a weakened inflammatory response, a patient's symptoms may temporarily improve, but not because the pathogenic bacteria which drive the Th1 diseases have been eradicated.
+Although the Th1 diseases are caused by bacterial pathogens, these alternatives to the Marshall Protocol are ineffective for at least several reasons. For one, antibiotics given in high enough doses interfere with immune activity. With a weakened inflammatory response, a patient's symptoms may temporarily improve, but not because the pathogenic bacteria which drive the Th1 diseases have been eradicated.
 Also, these protocols do not use olmesartan (Benicar) to activate the Vitamin D Receptor. Protocols which do not generate sustained immunopathology are ultimately not effective against the Th1 diseases.
 ===== Immunosuppression of high-dose antibiotics =====
@@ Line 127: / Line 128: @@
 ===== Notes and comments =====
-EDIT
   *Legacy content
     * f271
+<blockquote>Shifting the balance: antibiotic effects on host–microbiota mutualism Benjamin P. Willing1,3, Shannon L. Russell1,2,3 & B. Brett Finlay1,2  About the authors
+topof page
+Abstract
+Antibiotics have been used effectively as a means to treat bacterial infections in humans and animals for over half a century. However, through their use, lasting alterations are being made to a mutualistic relationship that has taken millennia to evolve: the relationship between the host and its microbiota. Host–microbiota interactions are dynamic; therefore, changes in the microbiota as a consequence of antibiotic treatment can result in the dysregulation of host immune homeostasis and an increased susceptibility to disease. A better understanding of both the changes in the microbiota as a result of antibiotic treatment and the consequential changes in host immune homeostasis is imperative, so that these effects can be mitigated.
+</blockquote>
 ===== References =====

home/othertreatments/antibacterials/highdose.txt · Last modified: 09.14.2022 by 127.0.0.1