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home:pathogenesis:evidence_bacteria [05.02.2011] – external edit 127.0.0.1 | home:pathogenesis:evidence_bacteria [01.13.2020] – [Other evidence] sallieq | ||
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- | The mainstream, but antiquated, view about chronic disease is best expressed by a certain physician thusly: "Of our thousand bacterial species, I only have to worry about a couple dozen." However, there is substantial evidence that chronic diseases are caused by pathogens as opposed to [[home: | + | The mainstream, but antiquated, view about chronic disease is best expressed by a certain physician thusly: "Of our thousand bacterial species, I only have to worry about a couple dozen" |
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+ | However, there is substantial evidence that chronic diseases are caused by pathogens as opposed to [[home: | ||
* researchers' | * researchers' | ||
* familial aggregation of disease and incidence in small communities in close contact | * familial aggregation of disease and incidence in small communities in close contact | ||
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In addition, it seems highly likely that supposedly non-infectious chronic diseases are in fact caused by pathogens when one considers their clinical features, histology, treatment response, microbe populations, | In addition, it seems highly likely that supposedly non-infectious chronic diseases are in fact caused by pathogens when one considers their clinical features, histology, treatment response, microbe populations, | ||
- | According to the [[home:: | + | According to the Marshall Pathogenesis, |
Besides the [[home: | Besides the [[home: | ||
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* **Treatment response** – Whatever the chronic inflammatory condition, patients on the Marshall Protocol invariably experience the tell-tale immunopathological reaction, which can only be described as a bacterial die-off reaction. Though a number of other antibacterial treatments are ultimately less effective than the MP, patients also respond to these. Even high-dose antibiotics, | * **Treatment response** – Whatever the chronic inflammatory condition, patients on the Marshall Protocol invariably experience the tell-tale immunopathological reaction, which can only be described as a bacterial die-off reaction. Though a number of other antibacterial treatments are ultimately less effective than the MP, patients also respond to these. Even high-dose antibiotics, | ||
* < | * < | ||
- | * **[[home: | + | * **[[home: |
[{{ : | [{{ : | ||
* **Failure of lifestyle interventions** – It has been widely hypothesized that lifestyle factors, including a poor diet and a lack of exercise, are the primary driver behind increased rates of chronic disease. For example, the World Health Organization [[http:// | * **Failure of lifestyle interventions** – It has been widely hypothesized that lifestyle factors, including a poor diet and a lack of exercise, are the primary driver behind increased rates of chronic disease. For example, the World Health Organization [[http:// | ||
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* **Disease appearing in scars** – There are several case reports of sarcoidosis lesions forming within scars, which are especially susceptible to infection. That these granuloma often take long periods of time to be realized corresponds with the growth rate of the slow-growing chronic pathogens which the Marshall Pathogenesis implicates in chronic disease.(({{pubmed> | * **Disease appearing in scars** – There are several case reports of sarcoidosis lesions forming within scars, which are especially susceptible to infection. That these granuloma often take long periods of time to be realized corresponds with the growth rate of the slow-growing chronic pathogens which the Marshall Pathogenesis implicates in chronic disease.(({{pubmed> | ||
* **Absence of an effect for most chemicals thought to be toxic** – In contrast to infectious agents, little evidence implicates typical doses of dietary chemicals as primary causes of human cancer. Paul Ewald, PhD has concluded that humans have evolved effective flexible enzymatic systems for degrading potentially carcinogenic chemicals.(({{pubmed> | * **Absence of an effect for most chemicals thought to be toxic** – In contrast to infectious agents, little evidence implicates typical doses of dietary chemicals as primary causes of human cancer. Paul Ewald, PhD has concluded that humans have evolved effective flexible enzymatic systems for degrading potentially carcinogenic chemicals.(({{pubmed> | ||
+ | * **Difficulty distinguishing autoimmune disease from infectious disease** – Although they have identified a signature that distinguishes healthy individuals from sarcoidosis or tuberculosis patients, the biosignatures of both diseases are nevertheless very similar. According to the [[http:// | ||
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If the common inflammatory diseases (including autoimmune diseases) were genetic, the only way they would manage not to be weeded out of the population would be if they conferred some sort of beneficial survival trait not related to the disease. | If the common inflammatory diseases (including autoimmune diseases) were genetic, the only way they would manage not to be weeded out of the population would be if they conferred some sort of beneficial survival trait not related to the disease. | ||
- | To date, no such benefits have been identified in any chronic disease, schizophrenia being a good example.(({{pubmed> | + | To date, no such benefits have been identified in any chronic disease, schizophrenia being a good example.(({{pubmed> |
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===== Notes and comments ===== | ===== Notes and comments ===== | ||
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* f127 | * f127 | ||
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+ | Sterile in health | ||
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+ | Characteristic of medically important bacteria' | ||
+ | --Review of Medical Microbiology and Immunology, 2008 | ||
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+ | </ | ||
===== References ===== | ===== References ===== |