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home:pathogenesis:evidence_bacteria [08.29.2012] – [Other evidence] paulalbert | home:pathogenesis:evidence_bacteria [01.13.2020] – [Other evidence] sallieq | ||
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===== Other evidence ===== | ===== Other evidence ===== | ||
- | There is also the following types of evidence, the first three of which are noted in a [[http:// | + | There is also the following types of evidence, the first three of which are noted in a [[http:// |
* **Clinical features** – Chronic diseases often share the same clinical presentation as those of known infectious diseases including fever and leucocytosis (an elevated number of white cells in the blood). | * **Clinical features** – Chronic diseases often share the same clinical presentation as those of known infectious diseases including fever and leucocytosis (an elevated number of white cells in the blood). | ||
* **Histology** – The inflammation of affected tissues in a chronic disease is very similar to inflammation caused by infection of characteristic microbial structures. [[http:// | * **Histology** – The inflammation of affected tissues in a chronic disease is very similar to inflammation caused by infection of characteristic microbial structures. [[http:// | ||
* **Treatment response** – Whatever the chronic inflammatory condition, patients on the Marshall Protocol invariably experience the tell-tale immunopathological reaction, which can only be described as a bacterial die-off reaction. Though a number of other antibacterial treatments are ultimately less effective than the MP, patients also respond to these. Even high-dose antibiotics, | * **Treatment response** – Whatever the chronic inflammatory condition, patients on the Marshall Protocol invariably experience the tell-tale immunopathological reaction, which can only be described as a bacterial die-off reaction. Though a number of other antibacterial treatments are ultimately less effective than the MP, patients also respond to these. Even high-dose antibiotics, | ||
* < | * < | ||
- | * **[[home: | + | * **[[home: |
[{{ : | [{{ : | ||
* **Failure of lifestyle interventions** – It has been widely hypothesized that lifestyle factors, including a poor diet and a lack of exercise, are the primary driver behind increased rates of chronic disease. For example, the World Health Organization [[http:// | * **Failure of lifestyle interventions** – It has been widely hypothesized that lifestyle factors, including a poor diet and a lack of exercise, are the primary driver behind increased rates of chronic disease. For example, the World Health Organization [[http:// | ||
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If the common inflammatory diseases (including autoimmune diseases) were genetic, the only way they would manage not to be weeded out of the population would be if they conferred some sort of beneficial survival trait not related to the disease. | If the common inflammatory diseases (including autoimmune diseases) were genetic, the only way they would manage not to be weeded out of the population would be if they conferred some sort of beneficial survival trait not related to the disease. | ||
- | To date, no such benefits have been identified in any chronic disease, schizophrenia being a good example.(({{pubmed> | + | To date, no such benefits have been identified in any chronic disease, schizophrenia being a good example.(({{pubmed> |