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home:pathogenesis:microbiota:lforms [08.23.2017] – [Survival mechanisms] sallieqhome:pathogenesis:microbiota:lforms [10.27.2019] – [Recent research] sallieq
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 Inflammation also drives many chronic conditions that are still classified as (noninfectious) autoimmune or immune-mediated (e.g., systemic lupus erythematosus, rheumatoid arthritis, Crohn’s disease). Both [the innate and adaptive immune systems] play critical roles in the pathogenesis of these inflammatory syndromes. Therefore, inflammation is a clear potential link between infectious agents and chronic diseases. Inflammation also drives many chronic conditions that are still classified as (noninfectious) autoimmune or immune-mediated (e.g., systemic lupus erythematosus, rheumatoid arthritis, Crohn’s disease). Both [the innate and adaptive immune systems] play critical roles in the pathogenesis of these inflammatory syndromes. Therefore, inflammation is a clear potential link between infectious agents and chronic diseases.
  
-Siobhán M. O'Connor of the Centers for Disease Control and Prevention [[http://www.cdc.gov/ncidod/Eid/vol12no07/06-0037.htm|concurs]], stating, “The epidemiologic, clinical, and pathologic features of many chronic inflammatory diseases are consistent with a microbial cause.” +Siobhán M. O'Connor of the Centers for Disease Control and Prevention [[https://wwwnc.cdc.gov/eid/article/12/7/06-0037_article|concurs]], stating, “The epidemiologic, clinical, and pathologic features of many chronic inflammatory diseases are consistent with a microbial cause.” 
-===== Future research =====+===== Recent research ===== 
 + 
 +L-form bacteria cohabitants in human blood: significance for health and diseases. (({{pubmed>long:28715646}})) 
 + 
 +Possible role of L-form switching in recurrent urinary tract infection  (({{pubmed>long:31558767}}))  
 + 
 +==== Future research ==== 
  
 There seems to be little incentive for scientists to study the L-form. Since the bacteria can be killed by simple low-dose antibiotic therapy, drug companies have little interest in investing money into related research. Researchers studying the L-form often find themselves with very little grant money but must still work long, tedious hours in the lab. There seems to be little incentive for scientists to study the L-form. Since the bacteria can be killed by simple low-dose antibiotic therapy, drug companies have little interest in investing money into related research. Researchers studying the L-form often find themselves with very little grant money but must still work long, tedious hours in the lab.
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 //**Gerald Domingue, PhD**// </blockquote> //**Gerald Domingue, PhD**// </blockquote>
  
 +[[https://www.the-scientist.com/news-opinion/directed-evolution-phage-display-nab-chemistry-nobel-64890|Directed evolution to shape the function of enzymes]], has received the Nobel Prize in Chemistry 2018.
  
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 +{{tag>pathogenesis Microbes_in_the_human_body}}
 ===== Notes and comments ===== ===== Notes and comments =====
 +
  
 Link removed  [[http://www.cancer.med.umich.edu/news/immune07.shtml|stated]]  Link removed  [[http://www.cancer.med.umich.edu/news/immune07.shtml|stated]] 
home/pathogenesis/microbiota/lforms.txt · Last modified: 09.14.2022 by 127.0.0.1
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