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home:pathogenesis:vitamind [08.22.2017] – [Notes and comments] sallieq | home:pathogenesis:vitamind [05.13.2019] – [Science behind vitamin D] sallieq | ||
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A number of studies have suggested that patients with chronic inflammatory diseases are deficient in 25-hydroxyvitamin D (25-D) and that consuming greater quantities of vitamin D, which further elevates 25-D levels, alleviates disease symptoms. | A number of studies have suggested that patients with chronic inflammatory diseases are deficient in 25-hydroxyvitamin D (25-D) and that consuming greater quantities of vitamin D, which further elevates 25-D levels, alleviates disease symptoms. | ||
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+ | <note important> | ||
+ | "The idea that widespread vitamin D deficiency exists in the world has never had any credibility, | ||
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+ | " | ||
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+ | "The mis-labeling of this compound as a vitamin is regrettable, | ||
Some years ago, molecular biology identified 25-D as a secosteroid. Secosteroids would typically be expected to depress inflammation, | Some years ago, molecular biology identified 25-D as a secosteroid. Secosteroids would typically be expected to depress inflammation, | ||
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{{section>: | {{section>: | ||
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+ | ==== Bioavailability ==== | ||
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+ | Impact of 25-hydroxyvitamin D, free and bioavailable fractions of vitamin D, and vitamin D binding protein levels on metabolic syndrome components | ||
==== Pregnancy ==== | ==== Pregnancy ==== | ||
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* [[http:// | * [[http:// | ||
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{{tag> | {{tag> | ||
===== Notes and comments ===== | ===== Notes and comments ===== | ||
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+ | //broken link// | ||
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* [[http:// | * [[http:// |