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home:protocol:olmesartan [11.27.2018] – [Other beneficial effects] sallieq | home:protocol:olmesartan [12.03.2018] – [Cardiovascular disease] sallieq | ||
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* inhibit liver fibrosis and aid liver healing(({{pubmed> | * inhibit liver fibrosis and aid liver healing(({{pubmed> | ||
* reduce insulin resistance in rats(({{pubmed> | * reduce insulin resistance in rats(({{pubmed> | ||
+ | * 6 mg/kg olmesartan reduces the inflammatory process and bone loss in rats(({{pubmed> | ||
* protect the mitochondria from age-associated damage from oxidation(({{pubmed> | * protect the mitochondria from age-associated damage from oxidation(({{pubmed> | ||
* play a protective role against proliferative diabetic retinopathy (({{pubmed> | * play a protective role against proliferative diabetic retinopathy (({{pubmed> | ||
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* act as an antiarrhythmic(({{pubmed> | * act as an antiarrhythmic(({{pubmed> | ||
* block the production of Angiotensin II, thus improving mortality rates in heart failure patients(({{pubmed> | * block the production of Angiotensin II, thus improving mortality rates in heart failure patients(({{pubmed> | ||
+ | * This study demonstrated that olmesartan reduced angiotensin II and aldosterone levels more effectively than azilsartan, resulting in a stable antihypertensive effect. Olmesartan also had an inhibitory effect on cardiac hypertrophy. Accordingly, | ||
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