This shows you the differences between two versions of the page.
Previous revision | |||
— | home:publications:benediktsson_autoimmunity_2010 [07.04.2022] (current) – external edit 127.0.0.1 | ||
---|---|---|---|
Line 1: | Line 1: | ||
+ | ====== Poster - Bipolar disorders and autoimmune disease share a similar etiology====== | ||
+ | |||
+ | |||
+ | **Type:** Conference presentation\\ | ||
+ | **Presenter: | ||
+ | **Conference: | ||
+ | **Location: | ||
+ | **Date: | ||
+ | **Additional Content: | ||
+ | |||
+ | |||
+ | |||
+ | |||
+ | |||
+ | ===== Abstract ===== | ||
+ | |||
+ | |||
+ | Since the mid 1800s we have known that some infectious agents can cause dramatic personality changes, for example in diseases such as syphilis, rabies, toxoplasmosis and neurologic lyme disease. Treatment was dominated for many years by psychological theorizing, much of it Freudian; biological approaches were essentially limited to genetics. Recently both persistent viruses and antibodies to pathogens have been detected in samples from patients with bipolar disease. “Autoantibodies” to brain proteins, nuclear material, and brain lipids, among others, have been detected in both schizophrenic and bipolar populations. Higher levels of interleukin (IL1, IL 2, IL 1 RA), CD 4, CD 8 and Th1 and Th2 cytokines have been found in bipolar patients before, during and after medical treatment, strongly suggesting that bipolar disorders and autoimmune disease share a similar etiology. | ||
+ | |||
+ | We have been conducting an observational study since 2002 of a VDR agonist along with pulsed low dose antibiotics for treatment of chronic inflammatory diseases. Intriguingly, | ||
+ | |||
+ | |||
+ | |||
+ | |||
+ | |||
+ | {{tag> | ||