This shows you the differences between two versions of the page.
Both sides previous revisionPrevious revisionNext revision | Previous revisionNext revisionBoth sides next revision | ||
home:social:stress [11.27.2010] – paulalbert | home:social:stress [04.10.2011] – [Notes and comments] paulalbert | ||
---|---|---|---|
Line 7: | Line 7: | ||
Not unlike vitamin D metabolism, the human stress response is governed by a sophisticated hormonal system, one which is also dysregulated in patients with chronic inflammatory diseases. Successful management of both stressors and a patient' | Not unlike vitamin D metabolism, the human stress response is governed by a sophisticated hormonal system, one which is also dysregulated in patients with chronic inflammatory diseases. Successful management of both stressors and a patient' | ||
- | Physical stress or trauma has also been accepted as a contributor for several chronic diseases | + | Physical stress or trauma has also been accepted as a partial |
+ | |||
===== Psychological stress and immune function ===== | ===== Psychological stress and immune function ===== | ||
Line 16: | Line 18: | ||
{{section>: | {{section>: | ||
- | |||
- | ===== Psychological stress and disease ===== | ||
+ | ===== Psychological stress and disease ===== | ||
- | Some researchers have concluded that stressors trigger disease. These researchers point to retrospective studies, which have found that a high proportion of certain kinds of patients reported uncommon emotional stress before disease onset.(({{pubmed> | + | Some researchers have concluded that stressors |
* periodontal disease (({{pubmed> | * periodontal disease (({{pubmed> | ||
* Graves' | * Graves' | ||
Line 110: | Line 111: | ||
*Legacy content | *Legacy content | ||
* http:// | * http:// | ||
+ | |||
+ | |||
+ | * explain car accidents, etc. as disease triggers1 2 Blaney: “I have observed in many patients that an important precipitator of increased Th1 disease is physical injury. My speculation is that the physical trauma further activated AT1 due to the wound response, suppressing the immune system further and allowing increased bacterial growth.” | ||
=====References===== | =====References===== |