ALS (Lou Gehrig's Disease)

Lou Gehrig was known as The Gentleman of Baseball. He played for the NY Yankees in the '20s and '30s. He had Amyotrophic Lateral Sclerosis (ALS) and, because of his fame and courage in fighting it, the disease also became known as Lou Gehrig's Disease. They made a movie about his life and his famous speech telling his beloved fans he had to quit.

Here is his bio.

A Washington Post columnist recently reviewed a new book about Lou Gehrig. He included an interesting quote from the book, “Luckiest Man: The Life and Death of Lou Gehrig” by Jonathan Eig, explaining that Gehrig's untimely death at age 39 from ALS changed it from an almost-unknown disease to a near-universally known disease.

Lida Mattman's slides of ALS blood show very aggressive CWD-bacteria. Dr. Mattman suggested that the bacteria causing ALS multiply rapidly and are, therefore, very aggressive. It isn't known if that translates into their being resistant to the MP or readily killed. Or if they are easily killed, if they would release more endotoxins. All these unknowns mean that treating ALS with the MP will have to be done very carefully. It is best in these situations for Dr to discuss the actual case with Dr Marshall.

Treatment with the MP

I personally believe that ALL the Th1 diseasesThe chronic inflammatory diseases caused by bacterial pathogens. result from the same bacterial pathogenesis. This belief is grounded in my understanding of how the bacteria directly drive the phagocytic biochemistry, causing the Th1 cytokineAny of various protein molecules secreted by cells of the immune system that serve to regulate the immune system. release.

The problem I have with discussing applicability of the MP to ALS, (or MS, or Diabetes, or even Parkinsons), is our current lack of understanding of how serious the immune system reactions might be, and in what form they might become manifest.

Consequently I try to focus on the diseases which we know respond well, and expand the list very slowly. I am always looking to link up with physicians who have these patients in a supportive atmosphere, preferably where 24/7 care is available from, for example, a mother/wife trained as a nurse, so that we can start to explore the outer limits of this Th1 pathogenesis.


There are so many competing forces pushing and tugging at ALS patients that it is hard for them to be compliant and just focus on their recovery. With ALS the physicians, and the community, know that the patient is dying, and will push Vitamin D and just about every other medication onto them in a futile attempt to slow the degradation. It is hard for the (sick) patient to focus on recovery in the face of such pressures.

All applicable diseases are treated in a similar fashion on the MP with a few exceptions. Because ALS usually has a rapid course, one should progress through the MP as rapidly as is tolerable. Neurological symptoms are targeted better by one antibiotic in the second phase so that may be a priority to use.

With more aggressive syndromes, like ALS, it becomes more important to quickly force down the 25-DThe vitamin D metabolite widely (and erroneously) considered best indicator of vitamin D "deficiency." Inactivates the Vitamin D Nuclear Receptor. Produced by hydroxylation of vitamin D3 in the liver. (we don't have a way of doing this yet) and of getting them onto the multiple antibiotic protocols ASAP. We don't really have a good way of doing that either. So there will be some small variation between optimal treatments, especially for the most serious Th1 syndromes.

Trevor Marshall, PhD

Evidence of infectious cause

Sample PubMed cite1)

Cell Wall-Deficient (CWD) Bacterial Pathogens: Could Amyotrophic Lateral Sclerosis (ALS) Be Due to One?

Authors: Koch, Arthur L. Source: Critical Reviews in Microbiology; Jul-Sep2003, Vol. 29 Issue 3, p215-221, 7p

Abstract: Recently, a number of diseases that had been thought previously to be caused by something other than an infectious agent are now known to be caused by bacteria. It now appears that it is not uncommon that bacteria, viruses, or fungi can cause diseases even when these organisms have not been detected or cultured. The most recent, well-publicized case is that of stomach ulcers; these are largely due to Helicobacter pylori infections. Here, the possibility is explored that amyotrophic lateral sclerosis (ALS) is caused by a cell wall-deficient microorganism.

Author Affiliations: 1Biology Department, Indiana University, Bloomington, IN 47405-6801, USA ISSN: 1040-841X Accession Number: 11262074

Minocycline Delays Disease Onset and Mortality in a Transgenic Model of ALS Neuropharmacology Vol.13 No 8 12 June 2002 Ludo Van Den Bosch, Petra Tilkin, Griet Lemmns and Wim Robberecht Laboratory of Neurology, Campus Gasthuisberg, Herestraat 49, B-3000, Leuven, Belium

Patient experiences

Active patients

Last updated: January 3, 2009

Patient interviews

  • there are no patient interviews with ALS
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Notes and comments



From: Scottk Date: 2009-11-27 20:53:34 Reply: http://www.marshallprotocol.com/reply.php?topic_id=12013

I continued benicar throughout. after a day of MMS I could straighten my toes which I could not before and I still can now that I have stopped MMS and resumed the full mp. My walking improved also but I am unsure if that was from stopping the abx/ip. so far the improvement in my walking has remained.

An interesting thing is that my photosensitivityAbnormal sensitivity to sunlight and bright lights. Also referred to as "sun flare" or "light flare." has returned so it is not the benicar alone that causes it in me, it is benicar and abx.

1,25d 46.25 pg/ml. 25D 14.8ng/ml.Ratio 3.125 02/08. D25 12.09ng/ml 02/09. ALS 2003, muscle wasting; neck, shoulders, arms, hands, dropfoot. Speech somewhat affected, breathing and swallowing affected very little.

- Scottk, our longest time member with ALS(?) recently dropped antibiotics and trialed chlorine dioxide (MMS) in conjunction with Benicar and reports a possible improvement in his toes. He has now returned to using just the MP again.

We don't have an official position on this anti-microbial specifically (http://mpkb.org/doku.php/home:othertreatments), but for an ALS patient, would his experimentation be considered a specially allowed case due to this disease having more of an 'acute infection' timeline?

It would seem that we should have some kind of written position?



Mysterious cells may play role in ALS


Although we found that the potential of these cells is more limited than previously thought, it might be possible to coax them to adopt different fates,“ Bergles says. “We only need to know what factors are restricting their development in the intact nervous system.

I wonder if these researchers even suspect that a pea-soup of microbiotaThe bacterial community which causes chronic diseases - one which almost certainly includes multiple species and bacterial forms. is probably responsible for the “mysterious” behavior of these cells?????


Vitamin D discovery outpaces FDA decision making.
Marshall TG
Bioessays30p173-82(2008 Feb)
home/diseases/als.txt · Last modified: 10.19.2018 by sallieq
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