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home:diseases:alzheimers_dementia [02.11.2019]
sallieq [Evidence of infectious cause]
home:diseases:alzheimers_dementia [08.12.2019]
sallieq [Other immune suppression]
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 According to the Marshall Pathogenesis,​ microbes subvert immune activity by suppressing expression of key nuclear receptors involved in innate immune activity such as the Vitamin D Receptor. The fact that over-production of amyloid suppresses the VDR suggests a role of VDR dysfunction in the Alzheimer'​s disease process. Indeed, a 2010 study in //Journal of Alzheimer'​s Disease// showed that the amyloids trigger neurodegeneration not only by inducing LVSCC A1C expression and NGF levels, but also by dramatically suppressing VDR expression.(({{pubmed>​long:​20966550}})) ​ According to the Marshall Pathogenesis,​ microbes subvert immune activity by suppressing expression of key nuclear receptors involved in innate immune activity such as the Vitamin D Receptor. The fact that over-production of amyloid suppresses the VDR suggests a role of VDR dysfunction in the Alzheimer'​s disease process. Indeed, a 2010 study in //Journal of Alzheimer'​s Disease// showed that the amyloids trigger neurodegeneration not only by inducing LVSCC A1C expression and NGF levels, but also by dramatically suppressing VDR expression.(({{pubmed>​long:​20966550}})) ​
  
-==== Other immune ​suppression ====+ 
 +Roles of Brain Angiotensin II in Cognitive Function and Dementia. ​ (({{pubmed>​long:​23304450}}))  
 + 
 +==== Immune ​suppression ====
  
  The most serious ​ health ​ endpoints ​ that  have  been  reported ​ to  be  associated ​ with  extremely ​ low  frequency ​ (ELF)  and/​or ​  The most serious ​ health ​ endpoints ​ that  have  been  reported ​ to  be  associated ​ with  extremely ​ low  frequency ​ (ELF)  and/​or ​
home/diseases/alzheimers_dementia.txt · Last modified: 08.12.2019 by sallieq
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