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home:diseases:anorexia_bulimia [08.12.2019] – [Alayne's strategy] sallieqhome:diseases:anorexia_bulimia [09.14.2022] (current) – external edit 127.0.0.1
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 Discussion in The Economist print edition 29 Sep 2005 Discussion in The Economist print edition 29 Sep 2005
-[[http://www.economist.com/displaystory.cfm?story_id=4455455|Molecular self-loathing]]+[[https://www.economist.com/displaystory.cfm?story_id=4455455|Molecular self-loathing]]
  
 ===== Evidence of infectious cause ===== ===== Evidence of infectious cause =====
  
- The intrinsic ability of bacteria to regulate their growth and to maintain their population within the gut suggests that gut bacteria can interfere with molecular pathways controlling energy balance in the host. (({{pubmed>long:27616451}}))+ The intrinsic ability of bacteria to regulate their growth and to maintain their population within the gut suggests that gut bacteria can interfere with molecular pathways controlling energy balance in the host. (({{pmid>long:27616451}}))
  
 ===== Immune overload ===== ===== Immune overload =====
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 An immune overload hypothesis would suggest that compensating withdrawal from some activities would help recovery from eating disorders, and certainly a reduction of unnecessary parental pressure may be needed. An immune overload hypothesis would suggest that compensating withdrawal from some activities would help recovery from eating disorders, and certainly a reduction of unnecessary parental pressure may be needed.
  
-[[http://www.eurekalert.org/pub_releases/2009-07/uoc--rpn072109.php|New imaging technology]] at U.Cal. San Diego provides insight into abnormalities in the brain circuitry of patients with anorexia nervosa that may contribute to the puzzling symptoms found in people with the eating disorder.+[[https://www.eurekalert.org/pub_releases/2009-07/uoc--rpn072109.php|New imaging technology]] at U.Cal. San Diego provides insight into abnormalities in the brain circuitry of patients with anorexia nervosa that may contribute to the puzzling symptoms found in people with the eating disorder.
 <blockquote>Under Kaye's leadership, the eating disorders treatment program at UC San Diego is developing treatments based on understanding the temperament and personality that drives anorexia, and helping patients to use such traits in constructive, rather then self-defeating ways.</blockquote>  <blockquote>Under Kaye's leadership, the eating disorders treatment program at UC San Diego is developing treatments based on understanding the temperament and personality that drives anorexia, and helping patients to use such traits in constructive, rather then self-defeating ways.</blockquote> 
  
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 So that is it. I am starting to feel normal again. I even have less of the nerve issues in my hands now. I didn't think things like that would ever go back to normal. But I can feel them normalizing. The word for me is beginning to be…NORMAL.  4.5 years on the Marshall, (was on one antibiotic protocol or another since July of 2005).  So that is it. I am starting to feel normal again. I even have less of the nerve issues in my hands now. I didn't think things like that would ever go back to normal. But I can feel them normalizing. The word for me is beginning to be…NORMAL.  4.5 years on the Marshall, (was on one antibiotic protocol or another since July of 2005). 
 +
 +
 +====  Alayne's strategy ====
 + 
 +The trick is to have as many kinds of foods around as possible because when one's appetite is down, having just one thing to choose from generally doesn't work. I went through years of having little or no appetite myself, so understand the problem pretty well myself - it's very frustrating! 
 +
 +Here's some of what Jack manages to choke down and hasn't lost anymore weight on:
 +
 +Breakfasts - 2 slices of whole grain toast or rolls with peanut butter/almond butter with agave nectar (like honey) and high fat cheese.
 +
 +Mixed whole grain hot cereals with half & half, dried or whatever kinds of fruits you have and nuts.
 +
 +Smoothies - Frozen and unfrozen fruits (lots of vitamins). He also adds almonds or some softer nuts to the blenderized drink. Always add some protein to the mix as well. We like to make an eggwhite omelet to accompany the smoothie (3-5 eggwhites with garlic, grated cheese, green onions, tomatoes…or whatever else is in the fridge).
 +
 +Other meals - sandwiches with guacamole and whole milk cheese or chicken or both. We also use vegenaise which is a great substitute for mayo (tastes like the real thing).
 +
 +Brown rice, wild rice, basmati rice pilafs with quick cooked organic beef or lamb stew with lots of veggies and spices.
 +
 +Buffalo, beef, or lamb burgers (spiced with various herbs and onion, garlic, etc.) on whole grain rolls or just with veggies.
 +
 +Grill a marinated beef or buffalo steak and have on hand for snacks and meals.
 +
 +Roast a chicken once or twice a week and have on hand for snacks and meals. We make a great chicken salad with the vegenaise, pickles, onions, and whatever else we like. Spread on crackers, bread, or use in salads lettuces.
 +
 +Whole fat cheese on crackers (try different types of cheeses and crackers for variation)
 +
 +Apples and celery with peanut butter or almond butter. Cashew butter's also great.
 +
 +Jacks makes almond milk and cashew yogurt which are nice and high in calories. If you're interested, can give you the recipes. Kind of a pain to make though.
 +
 +Lots of sugar free dark chocolate.
 +
 +Mixed nuts (I know you're heartily sick of them…there are ways to flavor them differently so they're a tad more interesting. Although it's not known whether or not pumpkin and sunflower seeds have Vit D, Jacks eats them on many things. His 25-D dropped to <5, so I think they probably have little D, if any.
 +
 +Tortilla chips with salsas and guacamole.
  
 ===== Further Research  ===== ===== Further Research  =====
  
-We found significantly altered correlations between alpha-MSH autoAb levels and the total Eating Disorder Inventory-2 score, as well as most of its subscale dimensions in AN and BN patients vs. controls. Remarkably, these correlations were opposite in AN vs. BN patients. In contrast, levels of autoAbs reacting with adrenocorticotropic hormone, OT, or VP had only few altered correlations with the Eating Disorder Inventory-2 subscale dimensions in AN and BN patients. Thus, our data reveal that core psychobehavioral abnormalities characteristic for eating disorders correlate with the levels of autoAbs against alpha-MSH, suggesting that AN and BN may be associated with autoAb-mediated dysfunctions of primarily the melanocortin system.  //Fetissov SO, et al//(({{pubmed>long:16195379}}))+We found significantly altered correlations between alpha-MSH autoAb levels and the total Eating Disorder Inventory-2 score, as well as most of its subscale dimensions in AN and BN patients vs. controls. Remarkably, these correlations were opposite in AN vs. BN patients. In contrast, levels of autoAbs reacting with adrenocorticotropic hormone, OT, or VP had only few altered correlations with the Eating Disorder Inventory-2 subscale dimensions in AN and BN patients. Thus, our data reveal that core psychobehavioral abnormalities characteristic for eating disorders correlate with the levels of autoAbs against alpha-MSH, suggesting that AN and BN may be associated with autoAb-mediated dysfunctions of primarily the melanocortin system.  //Fetissov SO, et al//(({{pmid>long:16195379}}))
  
-Hypovolemia-induced obesity and diabetes. (({{pubmed>long:19709692}})) Hypovolemia is a diminished volume of circulating blood in the body. Therefore is it obviously important to guard against dehydration.+Hypovolemia-induced obesity and diabetes. (({{pmid>long:19709692}})) Hypovolemia is a diminished volume of circulating blood in the body. Therefore is it obviously important to guard against dehydration.
  
-Imaging studies reveal[[http://www.eurekalert.org/pub_releases/2009-07/uoc--rpn072109.php|Differences]] in the brain circuitry of patients with anorexia nervosa (commonly known as anorexia) that may contribute to the puzzling symptoms found in people with the eating disorder.  (({{pubmed>long:21243469}}))+Imaging studies reveal[[https://www.eurekalert.org/pub_releases/2009-07/uoc--rpn072109.php|Differences]] in the brain circuitry of patients with anorexia nervosa (commonly known as anorexia) that may contribute to the puzzling symptoms found in people with the eating disorder.  (({{pmid>long:21243469}}))
  
  
-[[http://www.plosone.org/article/info:doi/10.1371/journal.pone.0007125|Monitoring Bacterial Community]] of Human Gut Microbiota Reveals an Increase in Lactobacillus in Obese Patients and Methanogens in Anorexic Patients (({{pubmed>long:19774074}})) +[[https://www.plosone.org/article/info:doi/10.1371/journal.pone.0007125|Monitoring Bacterial Community]] of Human Gut Microbiota Reveals an Increase in Lactobacillus in Obese Patients and Methanogens in Anorexic Patients (({{pmid>long:19774074}})) 
  
-Famine may affect the microbiome in a lasting way(({{pubmed>long:14696131}})) +Famine may affect the microbiome in a lasting way(({{pmid>long:14696131}})) 
-[[http://www.ncbi.nlm.nih.gov/pubmed/14696131|psychological traits in eating disorders]]+[[https://www.ncbi.nlm.nih.gov/pubmed/14696131|psychological traits in eating disorders]]
  
-in New Insights in Anorexia Nervosa (({{pubmed>long:27445651}})) +in New Insights in Anorexia Nervosa (({{pmid>long:27445651}})) 
 P. Gorwood, J. Epelbaum and colleagues assemble seven possible models, each accompanied by illustrative figures. P. Gorwood, J. Epelbaum and colleagues assemble seven possible models, each accompanied by illustrative figures.
  
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 +<nodisp>
 ===== Notes and comments ===== ===== Notes and comments =====
  
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   * legacy content   * legacy content
-  * [[http://www.marshallprotocol.com/view_topic.php?id=4243&forum_id=37&jump_to=39895#p39895]] s60+  * [[https://www.marshallprotocol.com/view_topic.php?id=4243&forum_id=37&jump_to=39895#p39895]] s60
    
  
  
 (//Paul and/or Amy// ??) I'm not sure I can completely tease out the significance of this study, but this sort of result suggests that famine may affect the microbiome in a lasting way: (//Paul and/or Amy// ??) I'm not sure I can completely tease out the significance of this study, but this sort of result suggests that famine may affect the microbiome in a lasting way:
-[[http://www.ncbi.nlm.nih.gov/pubmed/14696131|psychological traits in eating disorders]]+[[https://www.ncbi.nlm.nih.gov/pubmed/14696131|psychological traits in eating disorders]]
  
 Final point: IMO, we should all be careful not to define the chain of events. What if a disruption in the microbiome preceded anorexia and the anorexia "phenotype" was only expressed in certain cultures? Final point: IMO, we should all be careful not to define the chain of events. What if a disruption in the microbiome preceded anorexia and the anorexia "phenotype" was only expressed in certain cultures?
  
-===== References =====+===== References =====</nodisp> 
home/diseases/anorexia_bulimia.txt · Last modified: 09.14.2022 by 127.0.0.1
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