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home:diseases:depression [12.17.2018] – [Patient interviews] sallieqhome:diseases:depression [12.17.2018] – [Evidence of infectious cause] sallieq
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 [[https://www.newscientist.com/article/mg22129530-400-psychobiotics-how-gut-bacteria-mess-with-your-mind/|How gut bacteria mess with your mind]] [[https://www.newscientist.com/article/mg22129530-400-psychobiotics-how-gut-bacteria-mess-with-your-mind/|How gut bacteria mess with your mind]]
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 +[[https://onlinelibrary.wiley.com/doi/full/10.1111/nmo.12198|Melancholic microbes]]
  
 [[https://www.ncbi.nlm.nih.gov/pubmed/28736331|The gut microbiota plays an important role in modulating small RNAs that influence hippocampal gene expression, a process critical to hippocampal development.]](({{pubmed>long:28736331}})) [[https://www.ncbi.nlm.nih.gov/pubmed/28736331|The gut microbiota plays an important role in modulating small RNAs that influence hippocampal gene expression, a process critical to hippocampal development.]](({{pubmed>long:28736331}}))
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 In the research field of psychoneuroimmunology, accumulating evidence has indicated the existence of reciprocal communication pathways between nervous, endocrine and immune systems. In this respect, there has been increasing interest in the putative involvement of the immune system in psychiatric disorders. Cytokines and major depression (({{pubmed>long:15694227}}))  In the research field of psychoneuroimmunology, accumulating evidence has indicated the existence of reciprocal communication pathways between nervous, endocrine and immune systems. In this respect, there has been increasing interest in the putative involvement of the immune system in psychiatric disorders. Cytokines and major depression (({{pubmed>long:15694227}})) 
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 +===== Immune signaling =====
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 +A novel form of immune signaling revealed by transmission of the inflammatory mediator serotonin between dendritic cells and T cells (({{pubmed>long:16223770}}))  
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 ===== Co-morbidity ===== ===== Co-morbidity =====
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 ===== Management ===== ===== Management =====
  
-These preliminary results suggest that chronic depression, in contrast to acute melancholic depression, might be characterized by increased ACTH response to CRH challenge. Short-term treatment with vagus nerve stimulation was associated with normalization of this response. (({{pubmed>long:16005439}}))+==== How can I control my depression? ==== 
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 +Depression is common in Th1 diseases. It's important to recognize neuro symptoms as immunopathology so they can be managed to tolerable. See What is neuro immunopathology? 
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 +If you are having suicidal thoughts, tell someone immediately. 
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 +If your depression is intolerable, you should slow down immunopathology. See What to do when immunopathology (immune system reaction) is too strong. 
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 +Antidepressants 
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 +If slowing the immune response is ineffective, you may need to try an antidepressant. Many of the prescription antidepressant medications are compatible with the MP meds if depression is not resolved by reducing immunopathology.  
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 +Preliminary results suggest that chronic depression, in contrast to acute melancholic depression, might be characterized by increased ACTH response to CRH challenge. Short-term treatment with vagus nerve stimulation was associated with normalization of this response. (({{pubmed>long:16005439}}))
  
 Action of SSRIs  (({{pubmed>long:22251606}})) Action of SSRIs  (({{pubmed>long:22251606}}))
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-{{tag>diseases }}+
  
  
home/diseases/depression.txt · Last modified: 09.14.2022 by 127.0.0.1
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