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home:diseases:kidney_disease [02.17.2019] – [Kidney disease] sallieqhome:diseases:kidney_disease [02.19.2019] – [Patients experiences] sallieq
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-Am J Kidney Dis. 2009 Apr;53(4):711-4. Epub 2009 Jan 29. +Calcium-alkali syndrome due to vitamin D administration and magnesium oxide administration.(({{pubmed>long:19185403}}))
-Calcium-alkali syndrome due to vitamin D administration and magnesium oxide administration. +
-Hanada S, Iwamoto M, Kobayashi N, Ando R, Sasaki S. +
-Department of Nephrology, Musashino Red Cross Hospital, Tokyo Medical and Dental University, Tokyo, Japan. s-hanada@umin.ac.jp   (({{pubmed>long:19185403}}))+
  
  
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 Got Calcium? Welcome to the Calcium-Alkali Syndrome. Got Calcium? Welcome to the Calcium-Alkali Syndrome.
-Patel AM, Goldfarb S +
-J Am Soc Nephrol Apr 2010; +
-Download citation +
-Affiliation +
-Renal-Electrolyte and Hypertension Division, Department of Medicine, University of Pennsylvania School of Medicine, Philadelphia, Pennsylvania.+
 Abstract Abstract
 We recommend changing the name of the milk-alkali syndrome to the calcium-alkali syndrome, because the new terminology better reflects the shifting epidemiology and understanding of this disorder. The calcium-alkali syndrome is now the third most common cause of hospital admission for hypercalcemia, and those at greatest risk are postmenopausal or pregnant women.  We recommend changing the name of the milk-alkali syndrome to the calcium-alkali syndrome, because the new terminology better reflects the shifting epidemiology and understanding of this disorder. The calcium-alkali syndrome is now the third most common cause of hospital admission for hypercalcemia, and those at greatest risk are postmenopausal or pregnant women. 
  
-The incidence of the calcium-alkali syndrome is growing in large part as a result of the widespread use of over-the-counter calcium and vitamin D supplements.Advertising for treatment or prevention of osteoporosis has long encouraged this use. Intricate mechanisms mediating the calcium-alkali syndrome depend on interplay among intestine, kidney, and bone. New insights regarding its pathogenesis focus on the key role of calcium-sensing receptors and TRPV5 channels in the modulation of renal calcium excretion. Restoring extracellular blood volume, increasing GFR and calcium excretion, and discontinuing calcium supplementation provide best treatment.   (({{pubmed>long:20413609}}))+The incidence of the calcium-alkali syndrome is growing in large part as a result of the widespread use of over-the-counter calcium and vitamin D supplements. Advertising for treatment or prevention of osteoporosis has long encouraged this use.  
 + 
 +Intricate mechanisms mediating the calcium-alkali syndrome depend on interplay among intestine, kidney, and bone. New insights regarding its pathogenesis focus on the key role of calcium-sensing receptors and TRPV5 channels in the modulation of renal calcium excretion.  
 + 
 +Restoring extracellular blood volume, increasing GFR and calcium excretion, and discontinuing calcium supplementation provide best treatment.   (({{pubmed>long:20413609}}))
  
  
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-{{tag>disease kidneys arrange}}+{{tag>disease kidneys }}
  
  
home/diseases/kidney_disease.txt · Last modified: 09.14.2022 by 127.0.0.1
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