This shows you the differences between two versions of the page.
Both sides previous revisionPrevious revision | Next revisionBoth sides next revision | ||
home:starting:physician:reluctant [04.15.2019] – [Ascertain if reluctance is a provider problem or a bureaucracy problem] sallieq | home:starting:physician:reluctant [04.15.2019] – [Ascertain if reluctance is a provider problem or a bureaucracy problem] sallieq | ||
---|---|---|---|
Line 166: | Line 166: | ||
I expect some other countries also have centralised control, in which case patients may need to research | I expect some other countries also have centralised control, in which case patients may need to research | ||
+ | |||
+ | === Specific research === | ||
Line 172: | Line 174: | ||
OLM refs to print; | OLM refs to print; | ||
including information about dose relating to body weight, and dose timing | including information about dose relating to body weight, and dose timing | ||
- | Dose timing | ||
+ | |||
+ | Dose timing | ||
- | |||
short extract for each research study follows: | short extract for each research study follows: | ||
Line 195: | Line 197: | ||
cytokine damage (({{pubmed> | cytokine damage (({{pubmed> | ||
- | | + | in circadian rhythms between HR and MAP in CKD: Synchronization between the two rhythms was progressively lost as renal function deteriorated, |
- | renal protective effects of olmesartan may be better than those of other ARBs (({{pubmed> | + | |
- | olmesartan may uniquely increase urinary ACE2 level, which could offer additional renoprotective effects (({{pubmed> | + | renal protective effects of olmesartan may be better than those of other ARBs (({{pubmed> |
+ | |||
+ | olmesartan may uniquely increase urinary ACE2 level, which could offer additional renoprotective effects (({{pubmed> | ||
* treatment with olmesartan inhibited bone loss (({{pubmed> | * treatment with olmesartan inhibited bone loss (({{pubmed> | ||
Line 214: | Line 219: | ||
improvement of glycemic control & insulin resistance was only observed in olmesartan group (({{pubmed> | improvement of glycemic control & insulin resistance was only observed in olmesartan group (({{pubmed> | ||
OLM substantially delayed the development of left ventricular remodeling in type 2 diabetes (({{pubmed> | OLM substantially delayed the development of left ventricular remodeling in type 2 diabetes (({{pubmed> | ||
+ | |||
+ | |||
Long term treatment Data suggest 40 & 80 mg olmesartan are able to significantly remodel & destiffen the arterial wall material during long-term treatment, partly independently of blood pressure, compared with 20 mg. hyper.ahajournals.org/ | Long term treatment Data suggest 40 & 80 mg olmesartan are able to significantly remodel & destiffen the arterial wall material during long-term treatment, partly independently of blood pressure, compared with 20 mg. hyper.ahajournals.org/ | ||
+ | |||
+ | |||
===== Read more ===== | ===== Read more ===== | ||